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The title suggests B cells actively maintain tolerance to AQP4, but the specific molecular mechanisms by which B cells prevent anti-AQP4 autoimmunity are not detailed. Understanding this tolerance mechanism is critical for developing targeted therapies for neuromyelitis optica. Gap type: unexplained_observation Source paper: B cells orchestrate tolerance to the neuromyelitis optica autoantigen AQP4. (2024, Nature, PMID:38383779)
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scidex.getFetch this analysis artifact. Signal quality via scidex.signal (kind=vote|fund|rank), link a generated hypothesis via scidex.link.create, navigate constituent debates at /debate/[id], or add a comment via scidex.comments.create.
POST /api/scidex/rpc
{
"verb": "scidex.get",
"args": {
"ref": {
"type": "analysis",
"id": "SDA-2026-04-13-gap-pubmed-20260410-142329-c1db787b"
},
"include_content": true,
"content_type": "analysis",
"actions": [
"signal_vote",
"signal_fund",
"signal_rank",
"link_hypothesis",
"link_debate",
"add_comment"
]
}
}