Details

scope
cortex/HC
claim_text
Neuregulin 1 Type I Overexpression Is Associated with Reduced NMDA Receptor-Mediated Synaptic Signaling in Hippocampal Interneurons Expressing PV or CCK.
section_id
section_06
source_url
https://github.com/AllenNeuralDynamics/ComputationalReviewVIP/blob/95e761177f7d2ec565983d3307c14ec238f9677c/evidence/section_06_evidence_package.json
effect_size
20 Hz
review_repo
ComputationalReviewVIP
section_ref
wiki_page:computationalreviewvip-06-synaptic-properties
source_kind
review_finding
source_path
evidence/section_06_evidence_package.json
source_span
study_system
cortex/HC
section_title
Synaptic Properties and Connectivity
review_bundle_ref
analysis_bundle:ab-2ce40c33e827
replication_status
single
review_package_ref
analysis_bundle:ab-2ce40c33e827
source_artifact_ref
wiki_page:computationalreviewvip-06-synaptic-properties
origin_url
https://github.com/AllenNeuralDynamics/ComputationalReviewVIP/blob/95e761177f7d2ec565983d3307c14ec238f9677c/evidence/section_06_evidence_package.json
commit_sha
95e761177f7d2ec565983d3307c14ec238f9677c
created_by
persona-jerome-lecoq-gbo-neuroscience
repository_url
https://github.com/AllenNeuralDynamics/ComputationalReviewVIP
Raw fields (5)
raw_fields
{
  "n": "",
  "doi": "10.1523/eneuro.0418-17.2018",
  "claim": "Neuregulin 1 Type I Overexpression Is Associated with Reduced NMDA Receptor-Mediated Synaptic Signaling in Hippocampal Interneurons Expressing PV or CCK.",
  "title": null,
  "cite_key": "Kotzadimitriou2018",
  "evidence": "Neuregulin 1 Type I Overexpression Is Associated with Reduced NMDA Receptor-Mediated Synaptic Signaling in Hippocampal Interneurons Expressing PV or CCK.. Abstract Hypofunction of  N -methyl- d -aspartate receptors (NMDARs) in inhibitory GABAergic interneurons is implicated in the pathophysiology of schizophrenia (SZ), a heritable disorder with many susceptibility genes. However, it is still unclear how SZ risk genes interfere with NMDAR-mediated synaptic transmission in diverse inhibitory interneuron populations. One putative risk gene is neuregulin 1 ( NRG1 ), which signals via the receptor ",
  "effect_size": "20 Hz",
  "text_access": "fulltext",
  "study_system": "cortex/HC",
  "_source_cluster": "cluster_05_synaptic_connectivity",
  "replication_status": "single",
  "_source_cluster_index": 134,
  "claim_source_sentence": null,
  "replication_evidence_dois": []
}
source_refs
[
  "paper:paper-b6bbcb9d264a"
]
evidence_refs
[
  {
    "ref": "paper:paper-b6bbcb9d264a"
  }
]
source_policy
{
  "mode": "public_source_pointer_with_short_context",
  "notes": [
    "Local review repositories are read-only inputs.",
    "SciDEX stores paper metadata, structured evidence, file pointers, and short citation contexts; it does not copy full review prose."
  ],
  "source_commit_sha": "95e761177f7d2ec565983d3307c14ec238f9677c",
  "source_repository_url": "https://github.com/AllenNeuralDynamics/ComputationalReviewVIP"
}
evidence_summary
Neuregulin 1 Type I Overexpression Is Associated with Reduced NMDA Receptor-Mediated Synaptic Signaling in Hippocampal Interneurons Expressing PV or CCK.. Abstract Hypofunction of  N -methyl- d -aspartate receptors (NMDARs) in inhibitory GABAergic interneurons is implicated in the pathophysiology of schizophrenia (SZ), a heritable disorder with many susceptibility genes. However, it is still unclear how SZ risk genes interfere with NMDAR-mediated synaptic transmission in diverse inhibitory interneuron populations. One putative risk gene is neuregulin 1 ( NRG1 ), which signals via the receptor 

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