- raw_fields
{
"n": 0,
"doi": "10.3109/10673221003747609",
"claim": "Reduced NMDA neurotransmission on PV-containing cells may disturb developmental synaptic reorganization during late adolescence/early adulthood, contributing to schizophrenia onset via gamma oscillation deficits",
"evidence": "Reduced NMDA neurotransmission on PV-containing cells may disturb developmental synaptic reorganization during late adolescence/early adulthood, contributing to schizophrenia onset via gamma oscillation deficits",
"effect_size": null,
"text_access": "abstract_only",
"study_system": "review of human schizophrenia clinical data and animal models",
"replication_status": "replication_unknown",
"claim_source_sentence": "Evidence suggests that reduced glutamatergic neurotransmission via the N-methyl-D-aspartate (NMDA) receptors that are localized to inhibitory interneurons, perhaps especially the fast-spiking cells that contain the calcium-binding protein parvalbumin (PV), may contribute to gamma band synchrony deficits.",
"replication_evidence_dois": [],
"effect_size_source_sentence": null
}- source_refs
[
"paper:paper-7d82ca3822ba"
]
- source_span
Evidence suggests that reduced glutamatergic neurotransmission via the N-methyl-D-aspartate (NMDA) receptors that are localized to inhibitory interneurons, perhaps especially the fast-spiking cells that contain the calcium-binding protein parvalbumin (PV), may contribute to gamma band synchrony deficits.
- evidence_refs
[
{
"ref": "paper:paper-7d82ca3822ba"
}
]- source_policy
{
"mode": "public_source_pointer_with_short_context",
"notes": [
"Local review repositories are read-only inputs.",
"SciDEX stores paper metadata, structured evidence, file pointers, and short citation contexts; it does not copy full review prose."
],
"source_commit_sha": "df9fc7e8d455b084152c9d713558dae0013cef21",
"source_repository_url": "https://github.com/AllenNeuralDynamics/ComputationalReviewPV"
}