- raw_fields
{
"n": 0,
"doi": "10.3390/antiox14101154",
"claim": "Other findings suggest that nitric oxide (NO) is involved in the development of LID through a post-synaptic mechanism, reducing the L-DOPA-induced increase in ΔFosB, phospho-DARPP-32, and phospho-GluA1 α-amino-3-hydro...",
"cite_key": "Munoz2025b",
"evidence": "primary_experimental",
"effect_size": null,
"text_access": "fulltext",
"study_system": "",
"source_cluster_id": "cluster_09",
"replication_status": "replication_unknown",
"claim_source_sentence": "Other findings suggest that nitric oxide (NO) is involved in the development of LID through a post-synaptic mechanism, reducing the L-DOPA-induced increase in ΔFosB, phospho-DARPP-32, and phospho-GluA1 α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor subunit levels [ 57 , 58 ].",
"replication_evidence_dois": [],
"effect_size_source_sentence": null
}- source_refs
[
"paper:paper-b11a1005bd46"
]
- source_span
Other findings suggest that nitric oxide (NO) is involved in the development of LID through a post-synaptic mechanism, reducing the L-DOPA-induced increase in ΔFosB, phospho-DARPP-32, and phospho-GluA1 α-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor subunit levels [ 57 , 58 ].
- evidence_refs
[
{
"ref": "paper:paper-b11a1005bd46"
}
]- source_policy
{
"mode": "public_source_pointer_with_short_context",
"notes": [
"Local review repositories are read-only inputs.",
"SciDEX stores paper metadata, structured evidence, file pointers, and short citation contexts; it does not copy full review prose."
],
"source_commit_sha": "0632aae8abc141909207fe91f6349b9e36489c3b",
"source_repository_url": "https://github.com/AllenNeuralDynamics/ComputationalReviewLoops"
}