Details

scope
rodent striatum review
section_id
section_04
source_url
https://github.com/AllenNeuralDynamics/ComputationalReviewLoops/blob/0632aae8abc141909207fe91f6349b9e36489c3b/evidence/section_04_evidence_package.json
effect_size
review_repo
ComputationalReviewLoops
section_ref
wiki_page:computationalreviewloops-04
source_kind
review_finding
source_path
evidence/section_04_evidence_package.json
study_system
rodent striatum review
section_title
Striatal Output Pathways: Direct, Indirect, and Beyond
evidence_summary
review_bundle_ref
analysis_bundle:ab-d49e54403ef9
replication_status
replicated
review_package_ref
analysis_bundle:ab-d49e54403ef9
source_artifact_ref
wiki_page:computationalreviewloops-04
origin_url
https://github.com/AllenNeuralDynamics/ComputationalReviewLoops/blob/0632aae8abc141909207fe91f6349b9e36489c3b/evidence/section_04_evidence_package.json
commit_sha
0632aae8abc141909207fe91f6349b9e36489c3b
created_by
persona-jerome-lecoq-gbo-neuroscience
repository_url
https://github.com/AllenNeuralDynamics/ComputationalReviewLoops
Raw fields (6)
claim_text
Activity-dependent striatal synaptic plasticity at corticostriatal synapses is differentially regulated in direct- vs indirect-pathway SPNs and is altered in disease models, providing a substrate for procedural learning and a target for pathophysiology.
raw_fields
{
  "n": 0,
  "doi": "10.1016/j.neuron.2008.11.005",
  "claim": "Activity-dependent striatal synaptic plasticity at corticostriatal synapses is differentially regulated in direct- vs indirect-pathway SPNs and is altered in disease models, providing a substrate for procedural learning and a target for pathophysiology.",
  "cite_key": "Kreitzer2008",
  "evidence": "",
  "effect_size": "",
  "text_access": "fulltext",
  "study_system": "rodent striatum review",
  "source_cluster_id": "cluster_04",
  "replication_status": "replicated",
  "claim_source_sentence": "HFS-induced striatal LTD requires dopamine D2 receptors, G q -coupled group I mGluRs, L-type calcium channels, and CB1 receptor activation, but notably, not NMDA receptors or mAChRs ( Calabresi et al., 1992a ; Calabresi et al., 1997 ; Choi and Lovinger, 1997 ; Gerdeman et al., 2002 ; Kreitzer and Malenka, 2005 ; Sung et al., 2001 ).",
  "replication_evidence_dois": [
    "10.1146/annurev-neuro-061010-113641"
  ],
  "effect_size_source_sentence": null
}
source_refs
[
  "paper:paper-71f855d96201"
]
source_span
HFS-induced striatal LTD requires dopamine D2 receptors, G q -coupled group I mGluRs, L-type calcium channels, and CB1 receptor activation, but notably, not NMDA receptors or mAChRs ( Calabresi et al., 1992a ; Calabresi et al., 1997 ; Choi and Lovinger, 1997 ; Gerdeman et al., 2002 ; Kreitzer and Malenka, 2005 ; Sung et al., 2001 ).
evidence_refs
[
  {
    "ref": "paper:paper-71f855d96201"
  }
]
source_policy
{
  "mode": "public_source_pointer_with_short_context",
  "notes": [
    "Local review repositories are read-only inputs.",
    "SciDEX stores paper metadata, structured evidence, file pointers, and short citation contexts; it does not copy full review prose."
  ],
  "source_commit_sha": "0632aae8abc141909207fe91f6349b9e36489c3b",
  "source_repository_url": "https://github.com/AllenNeuralDynamics/ComputationalReviewLoops"
}

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