Version history

{
  "session_id": "debate-test-enrollment-protocol-6ecc51c9",
  "round_number": 6,
  "agent_persona": "persona-theorist",
  "agent_backend": "minimax-M2.7",
  "action": "debate",
  "content": "\n\n## Round 2: Theorist Response\n\n### Response to the Falsifier's Critique\n\nThe falsifier accuses my \"selective decline\" hypothesis of unfalsifiability—this charge misconstrues the nature of the claim. Selectivity is empirically testable. I propose specific, measurable predictions: (1) LTP at Schaffer collateral-CA1 synapses should show greater age-related impairment than LTP at mossy fiber-CA3 synapses due to differential calcium channel profiles; (2) NMDA receptor-dependent plasticity",
  "tokens_used": "0",
  "persona_id": "persona-domain_expert",
  "argument": "\n\n## Round 2: Theorist Response\n\n### Response to the Falsifier's Critique\n\nThe falsifier accuses my \"selective decline\" hypothesis of unfalsifiability—this charge misconstrues the nature of the claim. Selectivity is empirically testable. I propose specific, measurable predictions: (1) LTP at Schaffer collateral-CA1 synapses should show greater age-related impairment than LTP at mossy fiber-CA3 synapses due to differential calcium channel profiles; (2) NMDA receptor-dependent plasticity"
}