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SciDEX hypotheses

Hypothesis corpus

Scored, debated, and tradable. Each hypothesis carries composite, novelty, impact, mechanistic, druggability, and safety scores — plus a live market price when a collider is open.

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Disease: Alzheimer's diseaseParkinson's diseaseCancerDiabetesCardiovascular diseaseNeurodegenerationMultiple sclerosisAging
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Showing alzheimer hypotheses, sorted by Composite score.

  • Hippocampal-Cortical Transcriptomic Divergence Reveals Accelerated Neurodegeneration-Like Signatures

    CDKN2A alzheimer 52%

    p16INK4a+ senescent cell accumulation in hippocampus supports region-specific aging transcriptomics.

    h-aging-hippo-cortex-divergence
  • Myelin Breakdown-Amyloid Interaction Amplifies Cortical Aging-Neurodegeneration Overlap

    MBP alzheimer 51%
    58% posterior · 2 signals

    Epigenetic drift at MBP/PLP1 myelination genes supported by DNA methylation changes in aging mouse brain.

    h-aging-myelin-amyloid
  • ELF2-Mediated OPC Epigenetic Drift Drives Region-Dependent Myelin Dysfunction

    ELF2 alzheimer 51%
    53% posterior · 1 signal

    Progressive DNA methylation drift in aging mouse brain supports epigenetic mechanism for OPC dysfunction.

    h-aging-opc-elf2
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