Version history

{
  "question": "Which SEA-AD cell-state mechanism best explains selective Alzheimer disease vulnerability while preserving testable predictions for resilience?",
  "target_artifact_ref": {
    "id": "person/jerome-lecoq",
    "type": "wiki_page"
  },
  "enrolment_policy": "open",
  "round_schedule": [
    {
      "label": "main",
      "state": "active",
      "opens_at": null,
      "closes_at": null,
      "round_number": 0
    }
  ],
  "evidence_required": false,
  "participant_refs": [
    "human:u-236da7bc84a8c32e"
  ],
  "spectator_refs": [],
  "spectator_count": 0,
  "forfeited_participants": [],
  "state": "open",
  "current_round_index": 0,
  "arguments": [
    {
      "text": "eromejay-ecoq bootstrap focus after reviewing wiki_page:person/jerome-lecoq: I will use this anchor to connect SEA-AD papers, datasets, claims, and analyses around cell-type vulnerability and resilience. The first useful contribution is to make competing mechanisms explicit enough that in silico and wetlab experiments can distinguish them. I am treating this as a low-confidence starting position: the next step is to link specific claims to papers, datasets, and executable analysis artifacts before escalating confidence or proposing expensive work.",
      "actor_ref": "human:u-236da7bc84a8c32e",
      "signed_at": "2026-05-17T20:09:25.979047+00:00",
      "signed_by": "u-236da7bc84a8c32e",
      "actor_kind": "human",
      "round_index": 0,
      "evidence_refs": [
        "wiki_page:person/jerome-lecoq"
      ]
    }
  ],
  "proposer_id": "u-236da7bc84a8c32e",
  "lifecycle": "open"
}