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open_questions › What resolves this contention: Whether REM-sleep dysregulation in narcolepsy is driven primarily by peripheral/skin thermosensitivity (a top-down hypothalamic temperature sensing axis) or by an autonomous preoptic homeostatic generator. The two studies emphasise different control variables (thermal coupling vs. circuit-intrinsic homeostasis) for the same phenomenon. / Skin thermal dynamics and hypothalamic thermosensitivity dissociate REM sleep from cataplexy in narcolepsy, suggesting REM intrusion mechanisms are temperature-coupled. / Homeostatic regulation of REM sleep is centrally controlled by preoptic area neurons of the hypothalamus, framing REM rebound as a homeostatic process generated within preoptic circuitry. › diff

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