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30 results
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amyloid plaques, the β-amyloid load (volume percent) and the amyloid
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amyloid polyneuropathy (FAP ATTR Val30Met) cases unrelated to endemic foci
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amyloid clearance and to be most effective during sleep. Ablation
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amyloid-β. Immunotherapeutics aimed at reducing amyloid beta are in clinical
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amyloid precursor protein (APP) and presenilins (PS1, PS2) increase production
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amyloid hypothesis (Hardy and Selkoe, 2002Hardy J. Selkoe D.J. The amyloid
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amyloid β peptide, revealing a protective effect against amyloid-β and tau pathology
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amyloid plaques, respectively. Despite successful induction, amyloid disease-associated microglia
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Amyloid imaging using positron emission tomography (PET) has an emerging
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amyloid accumulation followed by tau-mediated neurodegeneration. Despite advances in anti
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amyloid-beta (Abeta) such that the Amyloid Cascade Hypothesis is the predominant
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amyloid and tau, but researchers are gradually moving away from
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amyloid-targeting drugs, the amyloid hypothesis needs to be reconsidered
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amyloid- and tau-PET. Amyloid-positive patients with EOAD had high
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amyloid-positive from amyloid-negative DLB cases (AUC = 0.75) and was associated
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- Paper An intranasally delivered peptide drug ameliorates cognitive decline in Alzheimer transgenic mice.
amyloid β (Aβ) peptides is considered to be the primary
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- Paper Accelerating rates of cognitive decline and imaging markers associated with β-amyloid pathology.
amyloid thresholds to include the range of amyloid associated with
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amyloid-β peptide (Aβ) isoform Aβ₄₀ or Aβ₄₂ contribute
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amyloid reduction and slowing of clinical decline in early AD. The objective
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Amyloid Precursor Protein (APP) into Amyloid-β and Microtubule Associated
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amyloid-β plaques expressed P2Y12 receptor at varying levels in amyloid
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amyloid pathology within the amygdala and significantly greater β-amyloid
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amyloid β and tau. Interestingly, the percentage of amyloid β targeting
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amyloid-β (Aβ) peptide. Donanemab, an antibody that targets a modified
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amyloid-β plaque and tau neurofibrillary tangle deposition, neuroinflammation is considered
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amyloid stitching of myelin sheaths. Amyloid fibrils appear to be an ideal
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amyloid β42 (Aβ42) and increased total tau proteins (T-Tau) and phosphorylated
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amyloid peptides that accumulate within the brain of individuals with
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amyloid β, suggesting that this pathway may remove amyloid β from
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amyloid peptides that accumulate in the brain of patients with
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