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30 results
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amyloid-β peptide (Aβ42) levels in cerebrospinal fluid or on amyloid
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Amyloid-β1-42 levels in cerebrospinal fluid or amyloid positron
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amyloid plaque drives pathogenesis of Alzheimer disease (AD). Reduction of amyloid
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amyloid β-positivity, referred to here as amyloid age, and in relation
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amyloid beta epitope that is present only in brain amyloid
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amyloid clearance. Change in amyloid, tau, and clinical decline after
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amyloid clearance. Conversely, APOE2 increased amyloid beta 1-42 (Aβ42
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amyloid-β plaques and neurofibrillary tangles. The amyloid hypothesis of Alzheimer
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amyloids. The general ability of amyloids to bind amyloid-specific
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amyloid wherever they encounter monomeric amyloid precursors. Amyloid proteins can also
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amyloids leading the way. Unlike disease-associated amyloids, functional amyloids
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- Paper Progression of cerebral amyloid angiopathy: accumulation of amyloid-beta40 in affected vessels.
amyloid (cerebral amyloid angiopathy, or CAA) are generally asymptomatic, but in advanced
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amyloid-positive and amyloid-negative older adults without cognitive impairment
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amyloid plaques, the β-amyloid load (volume percent) and the amyloid
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amyloid PET progression among initially amyloid PET negative (A-) individuals
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amyloids and, consequently, amyloid and virus research need to join
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amyloid (Aβ)42/Aβ40, as measured by a high-precision
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amyloid called curli during biofilm formation. The major curlin subunit
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amyloid peptide (Aβ). The β-amyloid peptide (Aβ) that results
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amyloid precursor protein. The compartmentation of the amyloid precursor protein
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amyloid-targeting therapy that resulted in robust amyloid plaque reduction
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amyloid plaque formation, the composition of beta-amyloid plaques, and the accompanying
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amyloid tracers bind to identical sites on amyloid-β fibrils
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amyloid angiopathy (CAA) is typified by the cerebrovascular deposition of amyloid
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Amyloid (Aβ) 1-42 ratio to predict amyloid positivity. To improve
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amyloid. In keeping with these findings, higher BACE1 protein and β-amyloid
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amyloid-β and tau), cerebrovascular (microinfarcts, macroinfarcts, and cerebral amyloid
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- Paper Biofilm-Associated Amyloid Proteins Linked with the Progression of Neurodegenerative Diseases.
amyloid proteins have emerged as significant contributors to the progression
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amyloid, so phagocytosis of amyloid plaques by microglia could be regulated
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amyloid polyneuropathy (FAP ATTR Val30Met) cases unrelated to endemic foci
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