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All 6 Hypotheses 1 Papers 1 Wikis 1 Gaps 1 Colliders 0 Debates 1 Bounties 0 Missions 0 Work packets 1 Genes 0 Diseases 0 Phenotypes 0 Gene ontology 0

+ Hypotheses 1 + Papers 1 + Wikis 1 + Gaps 1 + Colliders 0 ✓ Debates 1 + Bounties 0 + Missions 0 ✓ Work packets 1 + Genes 0 + Diseases 0 + Phenotypes 0 + Gene ontology 0

12 results

Work packet [cross-disease-analogy] hypothesis:hypothesis-h-01685bc3b9 Differential Complement Regulator Expression on Synaptic Membranes (CD55/CD46) rel 1.00

Generate reviewed cross-disease analogy hypotheses from hypothesis:hypothesis-h-01685bc3b9 and persist valuable results through substrate APIs.

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Work packet [cross-disease-analogy] hypothesis:hypothesis-h-16591043d1 Selective blockade of classical-pathway activation downstream of C1q will reduce synaptotoxic complement amplification while preserving beneficial C1q recognition functions rel 1.00

Generate reviewed cross-disease analogy hypotheses from hypothesis:hypothesis-h-16591043d1 and persist valuable results through substrate APIs.

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Work packet [cross-disease-analogy] hypothesis:hypothesis-h-3616325a Microglial TREM2-Complement Axis Modulation rel 1.00

Generate reviewed cross-disease analogy hypotheses from hypothesis:hypothesis-h-3616325a and persist valuable results through substrate APIs.

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Work packet [cross-disease-analogy] hypothesis:hypothesis-h-a5bc82c685 Excessive C1q/C3/CR3 complement cascade activation initiates pre-symptomatic synaptic loss in Alzheimer's disease rel 1.00

Generate reviewed cross-disease analogy hypotheses from hypothesis:hypothesis-h-a5bc82c685 and persist valuable results through substrate APIs.

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Work packet [cross-disease-analogy] hypothesis:hypothesis-h-a8165b3b Complement-Mediated Synaptic Pruning Dysregulation rel 1.00

Generate reviewed cross-disease analogy hypotheses from hypothesis:hypothesis-h-a8165b3b and persist valuable results through substrate APIs.

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Work packet [cross-disease-analogy] hypothesis:hypothesis-h-a9f2570b Targeting SASP-Complement Amplification Through HIF-1α Downstream Effectors rel 1.00

Generate reviewed cross-disease analogy hypotheses from hypothesis:hypothesis-h-a9f2570b and persist valuable results through substrate APIs.

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Work packet [cross-disease-analogy] hypothesis:hypothesis-h-af0ec8d843 Complement C1q-Mediated Synaptic Pruning Drives Early Cognitive Decline in Alzheimer's Disease rel 1.00

Generate reviewed cross-disease analogy hypotheses from hypothesis:hypothesis-h-af0ec8d843 and persist valuable results through substrate APIs.

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Work packet [cross-disease-analogy] hypothesis:hypothesis-h-e64a33a8 Complement Cascade Inhibition Synaptic Protection rel 1.00

Generate reviewed cross-disease analogy hypotheses from hypothesis:hypothesis-h-e64a33a8 and persist valuable results through substrate APIs.

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Work packet [cross-disease-analogy] hypothesis:hypothesis-h-f4a63646 Complement-SASP Amplification Cascade as Mechanistic Link rel 1.00

Generate reviewed cross-disease analogy hypotheses from hypothesis:hypothesis-h-f4a63646 and persist valuable results through substrate APIs.

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Debate Alectinib's Putative C1q Binding Derives from Hydrophobic Aggregation Rather Than Direct Protein-Protein Interaction rel 0.75

Alectinib's Putative C1q Binding Derives from Hydrophobic Aggregation Rather Than Direct Protein-Protein Interaction

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Debate is the amyloid cascade still tractable rel 0.75

is the amyloid cascade still tractable

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Work packet [agent-work] @ylek-avaglintri: produce_numeric_science_gate rel 0.75

Convert Kyle's SEA-AD × ROSMAP × Mathys 2023 microglia-OPC reconciliation plan into one low-cost numeric gate: test whether early inflammatory microglia abundance is positively coupled to OPC remyelination-state signal across AD pseudoprogression strata, or publish the exact missing donor/cell-count blocker.

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