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complement pathway. TSP-1 binds to complement proteins and inhibits
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- Paper Alzheimer's disease-associated complement gene variants influence plasma complement protein levels.
complement regulators clusterin (CLU) and complement receptor 1 (CR1), recognised
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complement in disease aetiology. Genetic variations in complement genes are widespread
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complement system influences this imbalance during psychosis onset. This study
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complement system, specifically the classical complement pathway, was associated with
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Complement fragments (opsonins; C1q, C3b and iC3b) interact with complement
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- Paper Messengers of coagulopathy: complement-carrying extracellular vesicles in SARS-CoV-2 infection.
complement-active phenotype. Sub-lytic complement attack, particularly membrane attack
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complement components, is pivotal. The complement system, known for its role
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complement factors in the alternative complement pathway were analyzed in the 3 groups
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complement system is responsible for our defense against pathogens. The complement
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complement pathway protein C1q and the alternative complement pathway activation
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- Paper The complement cascade in Alzheimer's disease: modern implications of an ancient immune protagonist.
complement activation and review the therapeutic potential of targeting complement
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complement function. As a result, complement 3a (C3a) and complement
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complement system, immune cells, and mediators of inflammation provides an efficient
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Complement, Inflammasome, and Microglial Crosstalk in Glaucoma: From Neurodegeneration to Immune
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complement by interacting with complement components. Here, we show that
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Complement overactivation mediates microglial synapse elimination in neurological diseases such
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complement factor H (CFH) activity, and the complement activation products
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complement system, long regarded as an arm of innate immunity
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Alectinib's Putative C1q Binding Derives from Hydrophobic Aggregation Rather Than Direct Protein-Protein Interaction
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is the amyloid cascade still tractable
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complement plays a critical role in controlling WNV infection in complement
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complement proteins, innate immune molecules, to these synaptic elements. We also
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complement cascade and impaired regulation of complement factors contribute to chronic
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complement system activation via the classical and lectin pathways. However
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complement activation pathway (especially C1q, r, s, and C4) are the strongest
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complement system plays crucial roles in a wide breadth of immune
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complement C3 with astrocytes was also observed. Overexpression of complement
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Complement and microglia mediate early synapse loss in Alzheimer mouse
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complement-mediated microglial synaptic elimination involved in neurocognitive dysfunction and anxiety
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