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30 results

Paper Manipulation of Mitochondria Dynamics Reveals Separate Roles for Form and Function in Mitochondria Distribution. rel 1.10

Mitochondria shape is controlled by membrane fusion and fission mediated

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Paper Increased calpain-1 in mitochondria induces dilated heart failure in mice: role of mitochondrial superoxide anion. rel 0.90

mitochondria, respectively. Transgenic up-regulation of mitochondria-targeted CAPN1 dose

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Paper Photoreceptor inner segments in monkey and human retina: mitochondrial density, optics, and regional variation. rel 0.90

mitochondria, inner segment dimensions, and optical properties, as a basis

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Paper PINK1 is selectively stabilized on impaired mitochondria to activate Parkin. rel 0.90

mitochondria to trigger their autophagy. How Parkin recognizes damaged mitochondria

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Paper Parvalbumin-Deficiency Accelerates the Age-Dependent ROS Production in Pvalb Neurons <i>in vivo</i>: Link to Neurodevelopmental Disorders. rel 0.70

mitochondria impairment in Pvalb neurons is discussed as an essential

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Paper Mouse lines with photo-activatable mitochondria to study mitochondrial dynamics. rel 0.60

mitochondria in tissues. To investigate mitochondrial dynamics in tissues and disease

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Paper Transfer of mitochondria from astrocytes to neurons after stroke. rel 0.60

mitochondria and transfer them to astrocytes for disposal and recycling

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Paper Targeting mitochondria in dermatological therapy: beyond oxidative damage and skin aging. rel 0.50

mitochondria in oxidative damage and skin aging has been a significant

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Paper Inhibition of mitochondria prevents cell death in kidney epithelial cells by intra- and extracellular acidification. rel 0.50

Mitochondria play a significant role in apoptosis and loss of their

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Paper Mitochondrial function in development and disease. rel 0.50

Mitochondria are organelles with vital functions in almost all eukaryotic

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Paper Parkin regulates amino acid homeostasis at mitochondria-lysosome (M/L) contact sites in Parkinson's disease. rel 0.50

mitochondria-lysosome contacts via destabilization of active Rab7. Subcellular metabolomics

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Paper Melatonin as a Guardian of Mitochondria: Mechanisms and Therapeutic Potential in Neurodegenerative Diseases. rel 0.50

mitochondria represent important therapeutic targets, effective interventions targeting mitochondrial function

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Paper Mitochondria-lysosome contact site dynamics and misregulation in neurodegenerative diseases. rel 0.50

mitochondria-lysosome membrane contact sites have been identified as a key pathway

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Paper New Insights Into the Role of Mitochondria Quality Control in Ischemic Heart Disease. rel 0.50

mitochondria (such as mitophagy, mtROS, and mitochondria-related inflammation) on IHD. In addition

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Paper Stress-induced hyperacetylation of microtubule enhances mitochondrial fission and modulates the phosphorylation of Drp1 at 616Ser. rel 0.50

Mitochondria dynamics results from fission and fusion events that may be unbalanced

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Paper Endoplasmic reticulum-mitochondria contacts: function of the junction. rel 0.40

mitochondria. Increased understanding is being gained of how ER-mitochondria

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Paper Developing a genetic approach to investigate the mechanism of mitochondrial competence for DNA import. rel 0.40

mitochondria are naturally competent for importing linear DNA. However, it has proven

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Paper Mitochondrial fission, fusion, and stress. rel 0.40

mitochondria when cells experience metabolic or environmental stresses. Fusion helps

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Paper Mitochondria and neuronal activity. rel 0.40

Mitochondria are central for various cellular processes that include ATP production

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Paper Mitochondrial dysfunction and oxidative stress in neurodegenerative diseases. rel 0.40

mitochondria have a central role in ageing-related neurodegenerative diseases

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Paper ALS-linked mutant SOD1 damages mitochondria by promoting conformational changes in Bcl-2. rel 0.40

mitochondria is thought to cause mitochondrial dysfunction. Whether mutant SOD1

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Paper PINK1 phosphorylates ubiquitin to activate Parkin E3 ubiquitin ligase activity. rel 0.40

mitochondria. However, it has been unclear how PINK1 activates and recruits

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Paper Mitochondria and ageing in Drosophila. rel 0.40

mitochondria as a by-product of energy metabolism. Consistent with

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Paper Proline transport in rat kidney mitochondria. rel 0.40

mitochondria was investigated both by using isotopic techniques and by spectroscopic

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Paper Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression. rel 0.30

mitochondria, and whether the presence of these species is associated

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Paper Conserved functions of Arabidopsis mitochondrial late-acting maturation factors in the trafficking of iron‑sulfur clusters. rel 0.30

mitochondria of all eukaryotes and additionally in plastids of photosynthetic

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Paper Ginsenoside Rb1 inhibits astrocyte activation and promotes transfer of astrocytic mitochondria to neurons against ischemic stroke. rel 0.30

mitochondria. Mitochondrial signal, mitochondrial membrane potential and ATP production detected

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Paper Emerging views of mitophagy in immunity and autoimmune diseases. rel 0.30

mitochondria. Accumulating evidence implicates elimination of dysfunctional mitochondria as a powerful

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Paper Symposium overview. rel 0.30

Mitochondria in cell life and cell death' held at the FASEB

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Paper Deregulated mitochondrial microRNAs in Alzheimer's disease: Focus on synapse and mitochondria. rel 0.30

mitochondria in the disease process; (5) latest research developments in synapse

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