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30 results

Paper Mitochondria transfer-based therapies reduce the morbidity and mortality of Leigh syndrome. rel 1.50

mitochondria to acceptor cells1-3. One possible consequence of mitochondria

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Paper Mitochondria-lysosome contacts regulate mitochondrial Ca(2+) dynamics via lysosomal TRPML1. rel 1.20

mitochondria, which was mediated by tethering of mitochondria-lysosome contact

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Paper Human iPSCs derived astrocytes rescue rotenone-induced mitochondrial dysfunction and dopaminergic neurodegeneration in vitro by donating functional mitochondria. rel 1.20

mitochondria with normal function. In addition, the half-life of mitochondria

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Paper Manipulation of Mitochondria Dynamics Reveals Separate Roles for Form and Function in Mitochondria Distribution. rel 1.10

Mitochondria shape is controlled by membrane fusion and fission mediated

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Paper Stress triggers gut dysbiosis via CRH-CRHR1-mitochondria pathway. rel 0.90

mitochondria respiration conferred resilience against stress-induced mitochondria damaging and epithelium

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Paper Photoreceptor inner segments in monkey and human retina: mitochondrial density, optics, and regional variation. rel 0.90

mitochondria, inner segment dimensions, and optical properties, as a basis

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Paper Autophagosomes form at ER-mitochondria contact sites. rel 0.90

mitochondria-associated ER membrane fraction under starvation conditions. Disruption of the ER-mitochondria

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Paper Presequence protease reverses mitochondria-specific amyloid-β-induced mitophagy to protect mitochondria. rel 0.90

mitochondria and has been shown to play a central role

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Paper Increased calpain-1 in mitochondria induces dilated heart failure in mice: role of mitochondrial superoxide anion. rel 0.90

mitochondria, respectively. Transgenic up-regulation of mitochondria-targeted CAPN1 dose

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Paper Optineurin is an autophagy receptor for damaged mitochondria in parkin-mediated mitophagy that is disrupted by an ALS-linked mutation. rel 0.90

mitochondria for degradation by autophagosomes. We examined the role of optineurin

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Paper Role of Mitochondria-ER Contact Sites in Mitophagy. rel 0.90

mitochondria-ER contact sites (MERCS). Upon damage, mitochondria produce reactive

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Paper PINK1 is selectively stabilized on impaired mitochondria to activate Parkin. rel 0.90

mitochondria to trigger their autophagy. How Parkin recognizes damaged mitochondria

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Paper The multifaceted role of mitochondria in autism spectrum disorder. rel 0.90

mitochondria. Failure to supply a sufficient amount of energy, seen

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Debate Is the amyloid cascade hypothesis still the best explanation for AD? rel 0.75

Is the amyloid cascade hypothesis still the best explanation for AD?

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Debate what is the root cause of alzheimer's rel 0.75

what is the root cause of alzheimer's

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Debate what is the root cause of alzheimer's rel 0.75

what is the root cause of alzheimer's

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Debate What molecular and cellular factors distinguish the subset of donors with severe cellular and molecular phenotypes who show steeper cognitive decline? rel 0.75

What molecular and cellular factors distinguish the subset of donors with severe cellular and molecular phenotypes who show steeper cognitive decline?

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Debate what role does microglial TREM2 play in Alzheimer disease pathology rel 0.75

what role does microglial TREM2 play in Alzheimer disease pathology

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Paper Fragmented mitochondria released from microglia trigger A1 astrocytic response and propagate inflammatory neurodegeneration rel 0.70

mitochondria into the neuronal milieu. The amount of damaged mitochondria

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Paper Super mitochondria-enriched extracellular vesicles enable enhanced mitochondria transfer. rel 0.70

Mitochondria transfer is a spontaneous process that releases functional mitochondria

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Paper Temporal dynamics of PARK2/parkin and OPTN/optineurin recruitment during the mitophagy of damaged mitochondria. rel 0.70

mitochondria downstream of PARK2, and induces autophagosome assembly around mitochondria

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Paper Neuroglial transmitophagy and Parkinson's disease. rel 0.70

mitochondria in 2-9 μ saccular structures (spheroids). Damaged mitochondria

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Paper Augmented microglial endoplasmic reticulum-mitochondria contacts mediate depression-like behavior in mice induced by chronic social defeat stress. rel 0.70

Mitochondria-associated membranes (MAMs) is a platform facilitating calcium transport

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Paper GAP43-dependent mitochondria transfer from astrocytes enhances glioblastoma tumorigenicity. rel 0.70

mitochondria between heterogeneous cell types has been confirmed in various

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Paper Fission and selective fusion govern mitochondrial segregation and elimination by autophagy. rel 0.70

mitochondria, individual mitochondria were photolabeled and tracked through fusion and fission

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Paper Kenny mediates the recruitment of the phagophore for ubiquitin-dependent mitophagy in Drosophila neurons. rel 0.70

mitochondria is essential to neuronal homeostasis. Mitophagy is a critical

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Paper GTPase dependent recruitment of Grif-1 by Miro1 regulates mitochondrial trafficking in hippocampal neurons. rel 0.70

mitochondria. Increasing Miro1 expression levels recruits Grif-1 to mitochondria

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Paper Nicorandil alleviates cardiac microvascular ferroptosis in diabetic cardiomyopathy: Role of the mitochondria-localized AMPK-Parkin-ACSL4 signaling pathway. rel 0.70

mitochondria-associated ferroptosis. Importantly, overexpression of mitochondria-localized AMPKα1 (mitoAα1

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Paper Mitochondria: An overview of their origin, genome, architecture, and dynamics. rel 0.70

Mitochondria are traditionally viewed as the powerhouses of eukaryotic cells

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Paper Parvalbumin-Deficiency Accelerates the Age-Dependent ROS Production in Pvalb Neurons <i>in vivo</i>: Link to Neurodevelopmental Disorders. rel 0.70

mitochondria impairment in Pvalb neurons is discussed as an essential

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