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30 results

Paper Ripks and Neuroinflammation. rel 0.80

neuroinflammation. Activation of RIPK1 promotes neuroinflammation. RIPK1 and RIPK3 are responsible

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Paper The role of innate immune genes in Alzheimer's disease. rel 0.70

neuroinflammation in Alzheimer's disease brain. SUMMARY: Targeting neuroinflammation via CD33

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Paper The role of Toll-like receptors and neuroinflammation in Parkinson's disease. rel 0.70

neuroinflammation in PD samples using postmortem human PD brains and various

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Paper Early Effects of Poly(I:C)-induced Neuroinflammation on Hippocampal Astrocyte Function and Glycolytic Metabolism. rel 0.50

neuroinflammation model to investigate its effects on astrocyte function and neurometabolic

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Paper Gastrodin regulates the TLR4/TRAF6/NF-κB pathway to reduce neuroinflammation and microglial activation in an AD model. rel 0.50

Neuroinflammation plays a crucial role in neurodegeneration. Research indicates that

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Paper Chitinase-3-like-1: a multifaceted player in neuroinflammation and degenerative pathologies with therapeutic implications. rel 0.50

neuroinflammation. It has emerged as a significant player in various

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Paper RORγt drives production of the cytokine GM-CSF in helper T cells, which is essential for the effector phase of autoimmune neuroinflammation. rel 0.40

neuroinflammation remains a subject of some debate, none of their

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Paper Artemisinin Attenuates Amyloid-Induced Brain Inflammation and Memory Impairments by Modulating TLR4/NF-κB Signaling. rel 0.40

neuroinflammation has become a new AD treatment strategy. Artemisinin has potent

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Paper Inhibition of EZH2 (Enhancer of Zeste Homolog 2) Attenuates Neuroinflammation via H3k27me3/SOCS3/TRAF6/NF-κB (Trimethylation of Histone 3 Lysine 27/Suppressor of Cytokine Signaling 3/Tumor Necrosis Factor Receptor Family 6/Nuclear Factor-κB) in a Rat Model of Subarachnoid Hemorrhage. rel 0.40

Neuroinflammation has been proven to play an important role in the pathogenesis

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Paper Lipocalin 2 induces neuroinflammation and blood-brain barrier dysfunction through liver-brain axis in murine model of nonalcoholic steatohepatitis. rel 0.40

neuroinflammation and neurodegeneration, but the mediators and critical pathways involved

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Paper EZH2 is involved in vulnerability to neuroinflammation and depression-like behaviors induced by chronic stress in different aged mice. rel 0.40

neuroinflammation in the central nervous system and subsequent behavioral impairment

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Paper Neuroinflammation in neurodegenerative disorders: the roles of microglia and astrocytes. rel 0.40

neuroinflammation and studies on therapeutic drugs that can modulate neuroinflammation

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Paper CD38 in Neurodegeneration and Neuroinflammation. rel 0.40

neuroinflammation. While CD38 is strongly expressed in brain cells including

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Paper Neuroinflammation in neurodegenerative disorders: the roles of microglia and astrocytes rel 0.40

neuroinflammation and studies on therapeutic drugs that can modulate neuroinflammation

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Paper TLR4-dependent neuroinflammation mediates LPS-driven food-reward alterations during high-fat exposure. rel 0.40

neuroinflammation in the dysregulation of the reward system following exposure

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Paper Rutin prevents tau pathology and neuroinflammation in a mouse model of Alzheimer's disease. rel 0.30

neuroinflammation, and neurodegeneration. Thus, targeting of tau pathology is expected

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Paper α-synuclein suppresses microglial autophagy and promotes neurodegeneration in a mouse model of Parkinson's disease. rel 0.30

neuroinflammation, which plays an important role in PD. However, the effect

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Paper Morroniside maintains mitochondrial homeostasis in microglia and mitigates neuroinflammation in experimental autoimmune encephalomyelitis. rel 0.30

neuroinflammation in the central nervous system is a hallmark of both

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Paper ZRMQ-22, a novel DYRK1A inhibitor, attenuates neuroinflammation and cognitive impairments in LPS-induced mice: a potential strategy for Alzheimer's disease. rel 0.30

neuroinflammation mouse model using C57BL/6 mice. These findings highlight ZRMQ

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Paper Microglial PD-1 stimulation by astrocytic PD-L1 suppresses neuroinflammation and Alzheimer's disease pathology. rel 0.30

neuroinflammation is a pathogenic component of Alzheimer's disease (AD) that

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Paper Betaine Mitigates Amyloid-β-Associated Neuroinflammation by Suppressing the NLRP3 and NF-κB Signaling Pathways in Microglial Cells. rel 0.30

neuroinflammation has been shown to be involved in the entire

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Paper Potential role of chitinase-3-like protein 1 (CHI3L1/YKL-40) in neurodegeneration and Alzheimer's disease. rel 0.30

neuroinflammation and disease diagnosis of Alzheimer's disease (AD). In the brain

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Paper Lomerizine inhibits LPS-mediated neuroinflammation and tau hyperphosphorylation by modulating NLRP3, DYRK1A, and GSK3α/β. rel 0.30

neuroinflammation, we investigated the effects of lomerizine on LPS-induced

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Paper Lomerizine inhibits LPS-mediated neuroinflammation and tau hyperphosphorylation by modulating NLRP3, DYRK1A, and GSK3α/β. rel 0.30

neuroinflammation, we investigated the effects of lomerizine on LPS-induced

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Paper MG53 ameliorates nerve injury induced neuropathic pain through the regulation of Nrf2/HO-1 signaling in rats. rel 0.30

neuroinflammation and oxidative stress. However, the relationship between MG53 and neuropathic

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Paper Lupeol restores dopaminergic function by suppressing glial activation in a Parkinson's disease mouse model. rel 0.30

neuroinflammation and loss of dopaminergic neurons. The neurotoxin MPTP (1-methyl

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Paper Aloe Polysaccharides against 3-Nitropropionic acid-induced Huntington's disease-like symptoms: Role of BDNF/NF-κB/Nrf2 signaling pathways. rel 0.30

neuroinflammation. Few effective treatments are available for Huntington's. Additionally

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Paper Early glycolytic reprogramming controls microglial inflammatory activation. rel 0.30

neuroinflammation plays an important role in the progression of neurodegenerative

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Paper Estradiol Prevents Amyloid Beta-Induced Mitochondrial Dysfunction and Neurotoxicity in Alzheimer's Disease via AMPK-Dependent Suppression of NF-κB Signaling. rel 0.30

neuroinflammation in disease progression. Aβ impairs mitochondrial function, which, in part

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Paper Interaction between autophagy and the NLRP3 inflammasome in Alzheimer's and Parkinson's disease. rel 0.30

neuroinflammation is a vital recognition receptor in innate cellular immunity

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