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8 results
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tau hyperphosphorylation, oligomerization, or propagation remain underdeveloped in the represented
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Does tau-directed lysosomal rescue alter microglial stress signaling?
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tau aggregation and synaptic loss remain undefined. Current anti-inflammatory
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No current trials investigate combinatorial approaches. Single-target therapies have
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tau pathology, amyloid burden, alpha-synuclein, TDP-43, and other
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Whether TBI accelerates amyloid aggregation, tau seeding, or neuroinflammation remains unresolved.
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tau pathology years later. Only one trial is actively investigating
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tau pathology, amyloid aggregation, and neuronal loss remain incompletely characterized
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