Astrocytes and Cortical Oscillations

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Astrocytes and Cortical Oscillations

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Source: https://github.com/AllenNeuralDynamics/ComputationalReviewAstrocytes/blob/1a55da0634a3bc04e5688792ed12141ce271d28e/content/10_oscillations.md

Citation anchors captured: 38

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  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...

  • 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference Three independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference. Ingiosi et al., combining head-fixed two-phot...

  • 4Citationpaper:paper-7f6000297c9cThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference Three independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference. Ingiosi et al., combining head-fixed two-phot...

  • 5Citationpaper:paper-0441c28ad125Three independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference Three independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference. Ingiosi et al., combining head-fixed two-phot...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference0 Three independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference1. Ingiosi et al., combining head-fixed two-phot...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference2 Three independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference3. Ingiosi et al., combining head-fixed two-phot...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference4 Three in vivo imaging studies of cortical astrocyte calcium across sleep and wake 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference5. Figure summarises qualitative directional convergence, not harmonised numerical effect sizes. Vaidyanathan et al.'s claim is a negative correlation of astrocyte calcium event rate with cortical slow-wave activity, not a direct amplitude comparison between...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference6 Three in vivo imaging studies of cortical astrocyte calcium across sleep and wake 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference7. Figure summarises qualitative directional convergence, not harmonised numerical effect sizes. Vaidyanathan et al.'s claim is a negative correlation of astrocyte calcium event rate with cortical slow-wave activity, not a direct amplitude comparison between...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference8 Three in vivo imaging studies of cortical astrocyte calcium across sleep and wake 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference9. Figure summarises qualitative directional convergence, not harmonised numerical effect sizes. Vaidyanathan et al.'s claim is a negative correlation of astrocyte calcium event rate with cortical slow-wave activity, not a direct amplitude comparison between...

  • 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference0 Correlational state differences leave open whether astrocytes drive or merely reflect cortical rhythm. Three causal experiments argue for a driving role on the slow oscillation, though each reports a different outcome and none shares a common effect metric (Figure {ref}fig:sec10-gpcr-slow-oscillation). Poskanzer and Yuste show that optogenetic activation of astrocytes in mouse visual cortex switches the local neur...

  • 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference1 Correlational state differences leave open whether astrocytes drive or merely reflect cortical rhythm. Three causal experiments argue for a driving role on the slow oscillation, though each reports a different outcome and none shares a common effect metric (Figure {ref}fig:sec10-gpcr-slow-oscillation). Poskanzer and Yuste show that optogenetic activation of astrocytes in mouse visual cortex switches the local neur...

  • 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference2 Correlational state differences leave open whether astrocytes drive or merely reflect cortical rhythm. Three causal experiments argue for a driving role on the slow oscillation, though each reports a different outcome and none shares a common effect metric (Figure {ref}fig:sec10-gpcr-slow-oscillation). Poskanzer and Yuste show that optogenetic activation of astrocytes in mouse visual cortex switches the local neur...

  • 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference3 Correlational state differences leave open whether astrocytes drive or merely reflect cortical rhythm. Three causal experiments argue for a driving role on the slow oscillation, though each reports a different outcome and none shares a common effect metric (Figure {ref}fig:sec10-gpcr-slow-oscillation). Poskanzer and Yuste show that optogenetic activation of astrocytes in mouse visual cortex switches the local neur...

  • 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference4 Correlational state differences leave open whether astrocytes drive or merely reflect cortical rhythm. Three causal experiments argue for a driving role on the slow oscillation, though each reports a different outcome and none shares a common effect metric (Figure {ref}fig:sec10-gpcr-slow-oscillation). Poskanzer and Yuste show that optogenetic activation of astrocytes in mouse visual cortex switches the local neur...

  • 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference5 Causal astrocyte GPCR/Ca2+ manipulations that alter cortical slow oscillations, grouped by direction of perturbation 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference6. Entries use different astrocyte perturbations (optogenetic activation, chemogenetic Gi vs. Gq, genetic IP3R2 knockout) and different outcome readouts (cortical LFP state, NREM depth and duration, polysomnographic...

  • 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference7 Causal astrocyte GPCR/Ca2+ manipulations that alter cortical slow oscillations, grouped by direction of perturbation 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference8. Entries use different astrocyte perturbations (optogenetic activation, chemogenetic Gi vs. Gq, genetic IP3R2 knockout) and different outcome readouts (cortical LFP state, NREM depth and duration, polysomnographic...

  • 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference9 Causal astrocyte GPCR/Ca2+ manipulations that alter cortical slow oscillations, grouped by direction of perturbation 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference0. Entries use different astrocyte perturbations (optogenetic activation, chemogenetic Gi vs. Gq, genetic IP3R2 knockout) and different outcome readouts (cortical LFP state, NREM depth and duration, polysomnographic...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference1 These in vivo cortical effects sit in visible tension with slice-based negative results. Agulhon et al., using the same IP3R2 line for loss of function and an MrgA1-Gq DREADD line for gain of function, conclude that neither increasing nor obliterating astrocytic calcium fluxes affects spontaneous or evoked excitatory synaptic transmission or plasticity in hippocampal CA1 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference2. This is the same prep...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference3 These in vivo cortical effects sit in visible tension with slice-based negative results. Agulhon et al., using the same IP3R2 line for loss of function and an MrgA1-Gq DREADD line for gain of function, conclude that neither increasing nor obliterating astrocytic calcium fluxes affects spontaneous or evoked excitatory synaptic transmission or plasticity in hippocampal CA1 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference4. This is the same prep...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference5 These in vivo cortical effects sit in visible tension with slice-based negative results. Agulhon et al., using the same IP3R2 line for loss of function and an MrgA1-Gq DREADD line for gain of function, conclude that neither increasing nor obliterating astrocytic calcium fluxes affects spontaneous or evoked excitatory synaptic transmission or plasticity in hippocampal CA1 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference6. This is the same prep...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference7 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference8 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 1Citationpaper:paper-aa304d547310Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference, 2Citationpaper:paper-4baefab2ada9Section {ref}sec:in-vivo-dynamics closed on a picture in which cortical astrocyte calcium is dominated by arousal and noradrenergic tone rather than by fine sensory drive [reitman2023natneurosci,ingiosi2022clocksamp]. A natural extension of that result is to ask whether state-dependent astrocyte activity also tracks — and causally influences — the oscillatory regimes that define cortical states. Three compleme...content/10_oscillations.md:line 4Open reference9 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference0 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference1 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference2 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference3 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference4 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference5 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference6 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference7 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference8 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference9 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference0 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference1 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference2 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

  • 3Citationpaper:paper-9a1be146dacdThree independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite2020natcommun]. Ingiosi et al., combining head-fixed two-phot...content/10_oscillations.md:line 8Open reference3 Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K+] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...

References

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  3. [bojarskaite_2020_natcommun] paper:paper-9a1be146dacd “Three independent in vivo imaging studies converge on the direction of the effect. Bojarskaite et al. report that cortical astrocyte calcium signals exhibit distinct features across the sleep–wake cycle and are reduced during sleep compared with wakefulness, using membrane-localised Lck-GCaMP6f in somatosensory cortex of freely behaving mice [bojarskaite_2020_natcommun]. Ingiosi et al., combining head-fixed two-phot...”
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  17. [zhang_2020_neuralregen] paper:paper-ee7d224ee700 “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  18. [roux_2015_jneurosci] paper:paper-8fbfa72e2c08 “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  19. [clasadonte_2017_neuron] paper:paper-13af8a0e5f02 “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  20. [mylvaganam_2014_frontphysiol] paper:paper-ee035ae3e552 “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  21. [vincze_2019_frontcell] paper:paper-7bbdf728a35a “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  22. [halassa_2009_neuron] paper:paper-752d0a3a1250 “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  23. [florian_2011_jneurosci] paper:paper-94f45ae78657 “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  24. [hines_2013_translpsychiatry] paper:paper-eb1a53222f37 “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  25. [schmitt_2012_jneurosci] paper:paper-d13c2e941ac6 “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  26. [zhou_2024_cellres] paper:paper-3a1eaf6c0b15 “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  27. [sakatani_2008_jneurosci] paper:paper-d9154e14559b “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”
  28. [lee_2014_procnatl] paper:paper-bf59badf906b “Three such indirect pathways are supported by the evidence. The first is extracellular potassium clearance through astrocytic Kir4.1 and the syncytium: when potassium buffering is compromised pharmacologically or in disease states, extracellular [K<sup>+</sup>] rises and neuronal excitability and paroxysmal oscillations change accordingly [rangroothrane_2013_natmed,bazhenov_2004_neurophysiology,onodera_2021_jneurosc...”

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