Cerebrovascular Physiology
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browse →- 01
Translational imaging studies of cortical spreading depression in experimental models for migraine aura.
cerebrovascular changes. The article finally explores prospects for physiological studies
- 02
Cerebrovascular protective functions of amyloid precursor protein: Progress and therapeutic prospects.
physiological conditions, amyloid precursor protein (APP) is critically important for normal brain development, neurogenesis, neuronal survival, and synaptic signaling. Dyshomeostasis of APP increases deposition and accumulation of amyloid
- 03
Effect of ferroptosis on chronic cerebral hypoperfusion in vascular dementia.
cerebrovascular disease and vascular risk factors. Chronic cerebral hypoperfusion (CCH) is a common pathological and physiological
- 04
EphrinB2-EphB4-RASA1 Signaling in Human Cerebrovascular Development and Disease.
Cerebrovascular Development and Disease. Zeng X(1), Hunt A(2), Jin SC(3), Duran D(2), Gaillard J(2), Kahle KT(4). Author information: (1)Department of Genetics, Yale School
- 05
The role of exercise in enhancing brain and cerebrovascular health via the bone-brain axis: implications for surgical and clinical interventions.
UNLABELLED: The bone–brain axis constitutes an interactive bidirectional signaling
- 06
Role of the blood-brain barrier in multiple sclerosis.
cerebrovascular abnormalities seen in MS brains and parallel the release of inflammatory cytokines. In this review we establish the importance of the role of the BBB in MS. Improvements
- 07
The role of JAK/STAT signaling pathway in cerebral ischemia-reperfusion injury and the therapeutic effect of traditional Chinese medicine: A narrative review.
cerebrovascular disease with symptoms caused by insufficient blood or oxygen supply to the brain. When blood supplied is restored after cerebral ischemia, secondary brain injury may occur, which is called
- 08
Single-photon emission tomography.
physiologic and pathologic features of brain neurobiology. It has been used for many years in diagnosis of several neurologic and psychiatric disorders. In this chapter, we discuss the current state
- 09
Loss of endothelial CD2AP causes sex-dependent cerebrovascular dysfunction.
cerebrovascular dysfunction. Vandal M(1), Institoris A(2), Reveret L(3), Korin B(4), Gunn C(1), Hirai S(1), Jiang Y(1), Lee S(1), Lee J(4), Bourassa
- 10
Apolipoprotein E and clusterin inhibit the early phase of amyloid-β aggregation in an in vitro model of cerebral amyloid angiopathy.
cerebrovascular amyloid-β (Aβ) deposition, which leads to lobar hemorrhage and dementia. Biological molecules affecting the development of CAA have not been fully characterized. In this study, we performed proteome
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