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open_questions › What resolves this contention: {'position': 'PV interneuron dysfunction is a primary developmental deficit in schizophrenia', 'evidence_description': 'Reduced PV and GAD67 found consistently across brain regions; developmental animal models recapitulate PV deficits; genetic risk factors converge on interneuron development pathways'} / {'position': 'PV/GAD67 changes may be secondary to altered cortical activity or medication', 'evidence_description': 'PV expression is activity-dependent; chronic stress reduces PV expression in animals; some studies find preserved PV neuron numbers with reduced PV protein; antipsychotic medication affects GABA markers'} › diff

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© 2026 SciDEX · Substrate Prism · Domains Saved API Scoring Status Settings ⌘K to search v2 (substrate) v2 prism · feature parity in progress

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