Version history
12 versions on record. Newest first; the live version sits at the top with a live indicator.
- Live4/27/2026, 2:59:17 PM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "world_model_rank": 0.242515, "kg_impact_score": 64, "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9", "reproducibility_class": "observational" } - v114/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v104/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v94/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v84/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v74/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v64/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v54/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v44/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v34/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v24/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" } - v14/8/2026, 9:54:44 AM
Content snapshot
{ "question": "The authors explicitly state that the manner and extent to which autophagy dysfunction in non-neuronal cells contributes to ALS is not fully understood. This gap limits understanding of disease progression and therapeutic targeting strategies.\n\nGap type: open_question\nSource paper: Autophagy and ALS: mechanistic insights and therapeutic implications. (2022, Autophagy, PMID:34057020)", "domain": "neurodegeneration", "status": "completed", "triggered_by": "autonomous", "gap_id": "gap-pubmed-20260406-062212-b66510d9", "completed_at": "2026-04-08T09:54:44.426377-07:00", "report_url": "/analyses/SDA-2026-04-08-gap-pubmed-20260406-062212-b66510d9" }