12 results for “Cerebrovascular Physiology”. Showing 12 of 39,449.
Translational imaging studies of cortical spreading depression in experimental models for migraine aura.
cerebrovascular changes. The article finally explores prospects for physiological studies
Cerebrovascular protective functions of amyloid precursor protein: Progress and therapeutic prospects.
physiological conditions, amyloid precursor protein (APP) is critically important for normal brain development, neurogenesis, neuronal survival, and synaptic signaling. Dyshomeostasis of APP increases deposition and accumulation of amyloid
Effect of ferroptosis on chronic cerebral hypoperfusion in vascular dementia.
cerebrovascular disease and vascular risk factors. Chronic cerebral hypoperfusion (CCH) is a common pathological and physiological
EphrinB2-EphB4-RASA1 Signaling in Human Cerebrovascular Development and Disease.
Cerebrovascular Development and Disease. Zeng X(1), Hunt A(2), Jin SC(3), Duran D(2), Gaillard J(2), Kahle KT(4). Author information: (1)Department of Genetics, Yale School
The role of exercise in enhancing brain and cerebrovascular health via the bone-brain axis: implications for surgical and clinical interventions.
UNLABELLED: The bone–brain axis constitutes an interactive bidirectional signaling
Role of the blood-brain barrier in multiple sclerosis.
cerebrovascular abnormalities seen in MS brains and parallel the release of inflammatory cytokines. In this review we establish the importance of the role of the BBB in MS. Improvements
The role of JAK/STAT signaling pathway in cerebral ischemia-reperfusion injury and the therapeutic effect of traditional Chinese medicine: A narrative review.
cerebrovascular disease with symptoms caused by insufficient blood or oxygen supply to the brain. When blood supplied is restored after cerebral ischemia, secondary brain injury may occur, which is called
Single-photon emission tomography.
physiologic and pathologic features of brain neurobiology. It has been used for many years in diagnosis of several neurologic and psychiatric disorders. In this chapter, we discuss the current state
Loss of endothelial CD2AP causes sex-dependent cerebrovascular dysfunction.
cerebrovascular dysfunction. Vandal M(1), Institoris A(2), Reveret L(3), Korin B(4), Gunn C(1), Hirai S(1), Jiang Y(1), Lee S(1), Lee J(4), Bourassa
Apolipoprotein E and clusterin inhibit the early phase of amyloid-β aggregation in an in vitro model of cerebral amyloid angiopathy.
cerebrovascular amyloid-β (Aβ) deposition, which leads to lobar hemorrhage and dementia. Biological molecules affecting the development of CAA have not been fully characterized. In this study, we performed proteome
APOE4 leads to blood-brain barrier dysfunction predicting cognitive decline.
Physiology and Neuroscience, Zilkha Neurogenetic Institute, Keck School of Medicine, University of Southern California, Los Angeles, CA, USA. zlokovic@usc.edu. (21)Alzheimer's Disease Research Center, Keck School of Medicine, University
Reconstruction of the Blood-Brain Barrier In Vitro to Model and Therapeutically Target Neurological Disease.
physiological component of the central nervous system (CNS), maintaining nutrients, clearing waste, and protecting the brain from pathogens. The inherent barrier properties of the BBB pose a challenge for therapeutic