25 results for “Stroke Therapy”. Showing 25 of 39,449.
Regulating astrocyte phenotype by Lcn2 inhibition toward ischemic stroke therapy.
stroke therapy. Lcn2 level is highly associated with the phenotypic
Brain repair mechanisms after cell therapy for stroke.
therapies hold great promise for brain repair after stroke. While
Nose-to-Brain Delivery of Circular RNA SCMH1-Loaded Lipid Nanoparticles for Ischemic Stroke Therapy.
Ischemic stroke represents one of the leading cerebrovascular diseases with
Mitochondria-containing extracellular vesicles from mouse vs. human brain endothelial cells for ischemic stroke therapy.
Ischemic stroke-induced mitochondrial dysfunction in the blood-brain barrier
Senescence and SASP Are Potential Therapeutic Targets for Ischemic Stroke.
stroke therapies have shown success in reducing mortality, post-stroke
Purinergic signaling in Stroke and Multiple Sclerosis: Prospects for therapies.
Stroke and multiple sclerosis involve oxygen and nutrient deprivation in the brain as well as inflammation, all of which lead to the release of glutamate and ATP. These compounds
Ischemic stroke and intracranial hemorrhage with aspirin, dabigatran, and warfarin: impact of quality of anticoagulation control.
therapy in Chinese patients with atrial fibrillation. We investigated the risks of ischemic stroke
Targeting the blood-brain barrier for the delivery of stroke therapies.
A variety of neuroprotectants have shown promise in treating ischemic
Brain Targeting Bacterial Extracellular Vesicles Enhance Ischemic Stroke Therapy via Efficient ROS Elimination and Suppression of Immune Infiltration.
Ischemic stroke (IS) as a detrimental neurological disease is accompanied
Percutaneous left atrial appendage occlusion in patients with non-valvular atrial fibrillation: implantation and up to four years follow-up of the AMPLATZER Cardiac Plug.
therapy. Ischaemic stroke was observed at an annual rate of 0.8% and the annual
Nanoparticles hijack calvarial immune cells for CNS drug delivery and stroke therapy.
The rapid accessibility of calvarial immune cells to the brain
Dabrafenib, an inhibitor of RIP3 kinase-dependent necroptosis, reduces ischemic brain injury.
treatment of melanoma, it might be repurposed for stroke therapy.
The Unconventional Growth Factors Cerebral Dopamine Neurotrophic Factor and Mesencephalic Astrocyte-Derived Neurotrophic Factor Promote Post-ischemic Neurological Recovery, Perilesional Brain Remodeling, and Lesion-Remote Axonal Plasticity.
they might help to identify targets for restorative stroke therapy.
Autophagy hub-protein p62 orchestrates oxidative, endoplasmic reticulum stress, and inflammatory responses post-ischemia, exacerbating stroke outcome.
regulator of I/R injury, which offers itself as target for stroke therapies.
Neurotoxicity of ischemic astrocytes involves STAT3-mediated metabolic switching and depends on glycogen usage.
reactive astrogliosis and suggest novel targets for restorative stroke therapy.
Dominant role of microglial and macrophage innate immune responses in human ischemic infarcts.
stroke therapy that targets pro-inflammatory microglia and macrophage activation
Molecular Pathogenesis of Ischemic and Hemorrhagic Strokes: Background and Therapeutic Approaches.
based on molecular mechanisms in addition to traditional stroke therapies.
HSYA attenuates Inflammation - Necroptosis to ameliorate secondary brain injury after intracerebral hemorrhage.
stroke therapy for centuries and is well known for its anti
Pterostilbene improves neurological dysfunction and neuroinflammation after ischaemic stroke via HDAC3/Nrf1-mediated microglial activation.
stroke therapy. CONCLUSION: The HDAC3/Nrf1 pathway regulates the stability and function
Neuroprotective mechanisms of cobalamin in ischemic stroke insights from network pharmacology and molecular simulations.
stroke therapy. SUPPLEMENTARY INFORMATION: The online version contains supplementary material
Unraveling the neuroprotective potential of scalp electroacupuncture in ischemic stroke: A key role for electrical stimulation.
neuroinflammation, and suggests avenues for optimized treatment strategies in stroke therapy.
Neutrophil Mobilization Triggers Microglial Functional Change to Exacerbate Cerebral Ischemia-Reperfusion Injury.
protein 14 may provide a novel therapeutic strategy for ischemic stroke therapy.
Tropism-shifted AAV-PHP.eB-mediated bFGF gene therapy promotes varied neurorestoration after ischemic stroke in mice.
stroke and evaluate its effectiveness in mediating basic fibroblast growth factor ( bFGF ) gene therapy
SIRT-1/RHOT-1/PGC-1α loop modulates mitochondrial biogenesis and transfer to offer resilience following endovascular stem cell therapy in ischemic stroke.
therapy in ischemic stroke holds a great promise. Past studies
Nose-to-brain delivery of stem cells in stroke: the role of extracellular vesicles.
therapy that can be administered days, weeks, or months after a stroke