25 results for “atherosclerosis”. Showing 25 of 39,449.
Asparagine endopeptidase cleaves apolipoprotein A1 and accelerates pathogenesis of atherosclerosis.
atherosclerosis. However, how AEP accelerates atherosclerosis development remains incompletely understood
TRAP1 drives smooth muscle cell senescence and promotes atherosclerosis via HDAC3-primed histone H4 lysine 12 lactylation.
atherosclerosis. As the role of TRAP1 in atherosclerosis remains elusive
The Association between Severity of Atherosclerosis and Lower Urinary Tract Function in Male Patients with Lower Urinary Tract Symptoms.
atherosclerosis group (8.75 ± 2.50 times, P < 0.05). Development of atherosclerosis
GLSP and GLSP-derived triterpenes attenuate atherosclerosis and aortic calcification by stimulating ABCA1/G1-mediated macrophage cholesterol efflux and inactivating RUNX2-mediated VSMC osteogenesis.
Atherosclerosis is the main pathophysiological foundation of cardiovascular disease, which
Imidazole propionate is a driver and therapeutic target in atherosclerosis.
Atherosclerosis is the main underlying cause of cardiovascular diseases. Its prevention
Myeloid-Specific Deletion of Epsins 1 and 2 Reduces Atherosclerosis by Preventing LRP-1 Downregulation.
Atherosclerosis by Preventing LRP-1 Downregulation. Brophy ML(1)(2), Dong
Integrated metabolomics and network pharmacology to reveal the mechanisms of Shexiang Baoxin pill against atherosclerosis.
atherosclerosis model was constructed to clarify the protective effect of SBP on atherosclerosis
The P2RY12 receptor promotes VSMC-derived foam cell formation by inhibiting autophagy in advanced atherosclerosis.
atherosclerosis. Pi S(1), Mao L(1), Chen J(1), Shi H(1), Liu Y(1), Guo X(1), Li Y(1), Zhou
Endothelial Cell-Derived Von Willebrand Factor, But Not Platelet-Derived, Promotes Atherosclerosis in Apolipoprotein E-Deficient Mice.
atherosclerosis. In vitro studies suggest that platelet-derived VWF (Plt-VWF) is biochemically
High expression of CASP1 induces atherosclerosis.
atherosclerosis remains unclear. The atherosclerosis dataset GSE132651 and GSE202625 profiles
Macrophage-Derived GSDMD Plays an Essential Role in Atherosclerosis and Cross Talk Between Macrophages via the Mitochondria-STING-IRF3/NF-κB Axis.
atherosclerosis. GSDMD (gasdermin D)-mediated pyroptosis is a programmed cell
Alternating high-fat diet enhances atherosclerosis by neutrophil reprogramming.
atherosclerosis initiation, progression and complications1. However, individuals often change their
Evaluation of the intraoperative specimens of the thoracic and abdominal aorta.
atherosclerosis and aortitis as well as their distribution in different
Ligilactobacillus Murinus and Lactobacillus Johnsonii Suppress Macrophage Pyroptosis in Atherosclerosis through Butyrate-GPR109A-GSDMD Axis.
atherosclerosis. Oral drugs such as aspirin have recently been found
APRIL limits atherosclerosis by binding to heparan sulfate proteoglycans.
atherosclerosis by binding to heparan sulfate proteoglycans. Tsiantoulas D(1), Eslami
Inflammation in atherosclerosis: pathophysiology and mechanisms.
Atherosclerosis imposes a heavy burden on cardiovascular health due to its indispensable
BMAL1-Downregulation Aggravates Porphyromonas Gingivalis-Induced Atherosclerosis by Encouraging Oxidative Stress.
atherosclerosis, but the underlying mechanisms are still elusive. OBJECTIVE: To elucidate
Differential effects of apolipoprotein A-I-mimetic peptide on evolving and established atherosclerosis in apolipoprotein E-null mice.
atherosclerosis) and vein-graft atherosclerotic lesions (evolving atherosclerosis) were assessed
Disruption of adipocyte HIF-1α improves atherosclerosis through the inhibition of ceramide generation.
Atherosclerosis is a chronic multifactorial cardiovascular disease. Western diets have
Genetic Variation in the Chemokine Network and Atherosclerosis Risk.
atherosclerosis. As the field moves towards a newer generation of atherosclerosis
Activation of Nrf2 inhibits atherosclerosis in ApoE-/- mice through suppressing endothelial cell inflammation and lipid peroxidation.
atherosclerosis is not explored. METHODS: RNA-sequencing and single-cell
TRIM47 Promotes Atherosclerosis by Activating NF-κB Signaling via IκBα Ubiquitination.
Atherosclerosis, a major contributor to cardiovascular diseases, is characterized by chronic
HEG1 Protects Against Atherosclerosis by Regulating Stable Flow-Induced KLF2/4 Expression in Endothelial Cells.
Atherosclerosis preferentially occurs in arterial regions of disturbed blood flow
ASIC1/RIP1 accelerates atherosclerosis via disrupting lipophagy.
Atherosclerosis, a major contributor to cardiovascular disease, remains a significant
NLRP3 Inflammasome and the IL-1 Pathway in Atherosclerosis.
atherosclerosis, the underlying pathology of cardiovascular diseases. Therefore, therapeutic targeting