25 results for “cardiac pathophysiology”. Showing 25 of 39,449.
Human cardiomyocyte progenitor cells differentiate into functional mature cardiomyocytes: an in vitro model for studying human cardiac physiology and pathophysiology.
To date, there is no suitable in vitro model to
Bend it like BIN1: how a membrane-curving adaptor protein shapes cardiac physiology.
cardiac aging, suggesting a bidirectional pathophysiology where both insufficient and excessive
Stroke Related Brain-Heart Crosstalk: Pathophysiology, Clinical Implications, and Underlying Mechanisms.
pathophysiological mechanisms contributing to these severe cardiac complications involve the hypothalamic
Cardiac challenges in patients with Fabry disease.
cardiac structures, but the underlying complex pathophysiologic mechanisms remain poorly
SGLT2 inhibitors: from glucose-lowering to cardiovascular benefits.
pathophysiological axes (i.e. at systemic, vascular, cardiac, and renal levels
Cardiac fibrosis induced by high-fat diet in ApoE-deficient male mice is exacerbated by genetic deletion of PACAP-PAC1 signaling.
cardiac disorders. Interstitial fibrosis has been identified as a central pathophysiological
Clinical Diagnosis, Imaging, and Genetics of Arrhythmogenic Right Ventricular Cardiomyopathy/Dysplasia: JACC State-of-the-Art Review.
cardiac death and heart failure. Our understanding of its pathophysiology
Diagnosis and management of cyanotic congenital heart disease: part I.
cardiac lesions in children, namely, tetralogy of Fallot, transposition of the great arteries and tricuspid atresia are reviewed. Pathology, pathophysiology
Vascular Cognitive Impairment and Dementia: Clinical Features, Neuropathology, and Biomarkers.
pathophysiology, the cardiovascular clinician is likely to encounter both in the clinic. Moreover, common cardiac
Expert consensus document: Mitochondrial function as a therapeutic target in heart failure.
pathophysiology of heart failure is complex, mitochondrial dysfunction seems to be an important target for therapy to improve cardiac
Targeting HDAC6 to treat heart failure with preserved ejection fraction in mice.
cardiac fibroblasts and enhances mitochondrial respiratory capacity in cardiomyocytes. In this work, our findings show that HDAC6 impacts on heart pathophysiology
New insights into the function and pathophysiology of the ectodomain sheddase A Disintegrin And Metalloproteinase 10 (ADAM10).
pathophysiology of ADAM10 that were previously poorly covered, such as the role of ADAM10 on extracellular vesicles, its contribution to virus entry, and its involvement in cardiac
Alcohol-induced gut microbial reorganization and associated overproduction of phenylacetylglutamine promotes cardiovascular disease.
pathophysiology. Fecal microbiota transplantation from pair-/alcohol-fed mice into naïve male mice demonstrates the transmissibility of PAGln production and the CVD phenotype. Independent of alcohol exposure, pharmacological-mediated increases
Alox15/15-HpETE Aggravates Myocardial Ischemia-Reperfusion Injury by Promoting Cardiomyocyte Ferroptosis.
Pathophysiology, Tianjin Medical University, China (W.C., L.L., X.S., Y.L., J.W., Z.C., D.A., Y.Z., X.X.). (2)Third Department of Breast Cancer, Tianjin Medical University Cancer Institute and Hospital, National Clinical Research
The role of vimentin, Connexin-43 proteins, and oxidative stress in the protective effect of propranolol against clozapine-induced myocarditis and apoptosis in rats.
pathophysiology. The present work aimed to study the mechanisms behind the protective effect of propranolol (PRO) against CLZ-induced myocarditis and the possible involvement of VIM and CX43. The effect
Cardiovascular response to thermoregulatory challenges.
cardiac diseases, kidney diseases, diabetes, and hypertension. Temperature-induced damage is thought to be related to enhanced sympathetic reactivity followed by activation of the sympathetic nervous system, renin-angiotensin system
Contemporary management of acute right ventricular failure: a statement from the Heart Failure Association and the Working Group on Pulmonary Circulation and Right Ventricular Function of the European Society of Cardiology.
pathophysiology, and of management approaches. Right ventricular mechanics and function are altered in the setting of either pressure overload or volume overload. Failure may also result from a primary reduction
Occurrence of Cardio-respiratory reflex during ophthalmic artery embolisation in the neurointervention radiology suite
pathophysiologic mechanism is not known and needs further research. Reactions usually occur following the insertion of the catheter into the cavernous segment of the ICA or during the cannulation
White Blood Cell Count and Major Adverse Cardiovascular Events After Percutaneous Coronary Intervention in the Contemporary Era: Insights From the PARIS Study (Patterns of Non-Adherence to Anti-Platelet Regimens in Stented Patients Registry).
cardiac death (HR, 1.10 [95% CI, 1.05-1.17]; P<0.001), and clinically indicated target revascularization (HR, 1.04 [95% CI, 1.00-1.09]; P=0.03) but not stent thrombosis
Placental senescence pathophysiology is shared between peripartum cardiomyopathy and preeclampsia in mouse and human.
pathophysiological process. However, what drives this process remains unclear. Using serum proteomics, we identified the senescence-associated secretory phenotype (SASP), a marker of cellular senescence associated with biological aging
Understanding ventriculo-arterial coupling.
cardiac function and the properties of the arterial system, or ventriculo-arterial (VA) coupling, provides then a comprehensive characterization of the performance of the cardiovascular system in both health
Anderson-Fabry cardiomyopathy: prevalence, pathophysiology, diagnosis and treatment.
Anderson-Fabry disease (AFD) is a lysosomal storage disease caused
Targeting Nrf2 with the triterpenoid CDDO-imidazolide attenuates cigarette smoke-induced emphysema and cardiac dysfunction in mice.
Chronic obstructive pulmonary disease (COPD), which comprises emphysema and chronic
Calcium Deregulation: Novel Insights to Understand Friedreich's Ataxia Pathophysiology.
Friedreich's Ataxia (FRDA) is a neurodegenerative disorder, characterized by
Limited oxygen in standard cell culture alters metabolism and function of differentiated cells.
pathophysiological states. Cell culture is often considered to be hyperoxic, but pericellular oxygen levels, which are affected by oxygen diffusivity and consumption, are rarely reported. Here, we provide evidence that