25 results for “copd”. Showing 25 of 39,449.
Identification of novel biomarkers related to neutrophilic inflammation in COPD.
COPD, and two genes were risk factors of COPD, which
Expression of Nitric Oxide Synthase Isoenzyme in Lung Tissue of Smokers with and without Chronic Obstructive Pulmonary Disease.
COPD smokers, and 15 smokers with COPD. Neuronal NOS (nNOS
The role of FGF-2 in smoke-induced emphysema and the therapeutic potential of recombinant FGF-2 in patients with COPD.
COPD remains limited. The mechanism of FGF2 in a COPD
Incidence of Chronic Obstructive Pulmonary Disease in People With Human Immunodeficiency Virus and Their Parents and Siblings in Denmark.
COPD). In matched cohort studies, we examined risk factors for COPD
Identification and experimental validation of PYCARD as a crucial PANoptosis-related gene for immune response and inflammation in COPD.
COPD mice. PYCARD was a critical PANoptosis-related gene in all COPD
Does chronic obstructive pulmonary disease with or without type 2 diabetes mellitus influence the risk of lung cancer? Result from a population-based cohort study.
COPD group than in the non-COPD group, with adjusted
Targeting Type 2 Inflammation and Epithelial Alarmins in Chronic Obstructive Pulmonary Disease: A Biologics Outlook.
COPD, 20-40% of patients with COPD exhibit type 2 inflammation
Targeting CCL2-CCR2 signaling pathway alleviates macrophage dysfunction in COPD via PI3K-AKT axis.
COPD. It enhanced our understanding of COPD's molecular mechanisms
High-intensity interval training alleviates COPD-induced gastrocnemius muscle dysfunction via the BRD4/PGC-1α axis through restoring mitochondrial function and oxidative fiber composition.
COPD preliminarily. COPD rat models were established using cigarette smoke
Increased airway iron parameters and risk for exacerbation in COPD: an analysis from SPIROMICS.
COPD and in smokers without COPD when compared to non-smoker
COPD exacerbations: finally, a more than ACCEPTable risk score
COPD) with mild/moderate airflow limitation and low exacerbation risk. This
Oxidised IL-33 drives COPD epithelial pathogenesis via ST2-independent RAGE/EGFR signalling complex.
COPD). Interleukin (IL)-33 released from damaged airway epithelia causes
PRKN-regulated mitophagy and cellular senescence during COPD pathogenesis.
COPD) pathogenesis. Mitophagy plays a crucial role in eliminating damaged
PRKN-regulated mitophagy and cellular senescence during COPD pathogenesis.
COPD) pathogenesis. Mitophagy plays a crucial role in eliminating damaged
RIPK1 kinase-dependent inflammation and cell death contribute to the pathogenesis of COPD.
COPD pathogenesis. We aimed to define the contribution of RIPK1
COPD exacerbations: finally, a more than ACCEPTable risk score.
COPD) with mild/moderate airflow limitation and low exacerbation risk. This
Therapeutic effect of phycocyanin on chronic obstructive pulmonary disease in mice.
COPD. OBJECTIVE: As inflammation and oxidative stress can aggravate COPD
Targeting accelerated pulmonary ageing to treat chronic obstructive pulmonary disease-induced neuropathological comorbidities.
COPD) is a major incurable health burden, ranking as the third
Bufei Yishen formula protects the airway epithelial barrier and ameliorates COPD by enhancing autophagy through the Sirt1/AMPK/Foxo3 signaling pathway.
COPD symptoms and suppresses airway epithelial inflammation. In this study
Primary Care Provider Experience With Proactive E-Consults to Improve COPD Outcomes and Access to Specialty Care.
COPD (InCasE), that improved patient outcomes after hospitalization for COPD
Telomere dysfunction causes sustained inflammation in chronic obstructive pulmonary disease.
COPD. METHODS: Prospective comparison of patients with COPD and age- and sex-matched
Sea buckthorn extract mitigates chronic obstructive pulmonary disease by suppression of ferroptosis via scavenging ROS and blocking p53/MAPK pathways.
COPD) requires further clarification. AIM OF THE STUDY: To elucidate
Mechanism of Action of the Plateau-Adapted Gene PPARA in COPD.
COPD) is a complex respiratory disorder influenced by various factors
Exacerbating factors induce different gene expression profiles in peripheral blood mononuclear cells from asthmatics, patients with chronic obstructive pulmonary disease and healthy subjects.
COPD) and severe asthma differ with regard to their causative
Targeting interleukin-33 and thymic stromal lymphopoietin pathways for novel pulmonary therapeutics in asthma and COPD.
COPD cohorts with non-T2 inflammation providing support. Currently available