25 results for “glioblastoma”. Showing 25 of 39,449.
High CD133 Expression Is Associated with Worse Prognosis in Patients with Glioblastoma.
glioblastoma. Ten studies with a total of 715 glioblastoma patients
Principles in the Management of Glioblastoma.
glioblastoma. Several typical genetic and epigenetic alterations in glioblastoma include
Characterizing and targeting glioblastoma neuron-tumor networks with retrograde tracing.
glioblastoma progression, offering a viral strategy to tackle glioblastoma. Together
Mitochondrial Calcium Uniporter Links Acetyl-CoA Metabolism and H3K27 Acetylation to Maintain Glioblastoma Stem Cells.
Glioblastoma stem cells (GSC) exhibit remarkable metabolic and epigenetic adaptability
Glioblastoma-instructed astrocytes suppress tumour-specific T cell immunity.
Glioblastoma is the most common and aggressive primary brain cancer
Astrocyte-Glioblastoma Stem Cell Interactions via Extracellular Vesicles Contribute to Distinct Vascular Structures.
Glioblastoma (GBM) is a highly malignant astrocytic tumor characterized by marked
Infiltrating plasma cells maintain glioblastoma stem cells through IgG-Tumor binding.
Glioblastoma is a highly aggressive primary brain tumor with glioblastoma
An update on the molecular biology of glioblastoma, with clinical implications and progress in its treatment.
Glioblastoma multiforme (GBM) is the most aggressive and common malignant
Myosin IIA suppresses glioblastoma development in a mechanically sensitive manner.
glioblastoma to disperse through the brain contributes to its lethality
Macrophage-mediated myelin recycling fuels brain cancer malignancy.
glioblastoma in the brain, are particularly reliant on crosstalk with
Glioblastoma and brain connectivity: the need for a paradigm shift.
glioblastoma prognosis remains bleak. The emerging field of cancer neuroscience
A miR-297/hypoxia/DGK-α axis regulating glioblastoma survival.
glioblastoma, patient prognosis remains disappointing. This failure in treatment has been
Fitness Screens Map State-Specific Glioblastoma Stem Cell Vulnerabilities.
Glioblastoma (GBM) is the most common and lethal primary brain
Hypoxia Increases Connexin46 and Connexin43 Levels in KNS-42 Glioblastoma Cells.
Glioblastoma multiforme is a devastating brain tumor that frequently progresses
Spatially resolved multi-omics deciphers bidirectional tumor-host interdependence in glioblastoma.
Glioblastomas are malignant tumors of the central nervous system hallmarked
Sanggenol L Enhances Temozolomide Drug Sensitivity by Inhibiting Mitophagy and Inducing Apoptosis Through the Regulation of the TRIM16-OPTN Axis in Glioblastoma.
Glioblastoma (GBM) is the most aggressive and lethal form of glioma
Adeno-associated virus delivered CXCL9 sensitizes glioblastoma to anti-PD-1 immune checkpoint blockade.
glioblastoma cells evade immunological detection, underscoring the need for strategic
GENO-32AN ANATOMIC TRANSCRIPTIONAL ATLAS OF GLIOBLASTOMA
glioblastoma is highly complex and its relationship to histologic features
10.1016/j.cell.2019.06.024
glioblastoma. We find that malignant cells in glioblastoma exist in four
Inhibition of PERK-mediated unfolded protein response acts as a switch for reversal of residual senescence and as senolytic therapy in glioblastoma.
Glioblastoma due to recurrence is clinically challenging with 10-15 months
A Patient-Derived Glioblastoma Organoid Model and Biobank Recapitulates Inter- and Intra-tumoral Heterogeneity.
Glioblastomas exhibit vast inter- and intra-tumoral heterogeneity, complicating the development
Circulating extracellular vesicles as biomarker for diagnosis, prognosis, and monitoring in glioblastoma patients.
glioblastoma patients. Plasma samples were collected from glioblastoma patients at multiple
Efficacy of Adding Veliparib to Temozolomide for Patients With MGMT-Methylated Glioblastoma: A Randomized Clinical Trial.
glioblastoma is poor following standard therapy with surgical resection, radiation
An anatomic transcriptional atlas of human glioblastoma
Glioblastoma is the most lethal form of human brain cancer
Cranioencephalic functional lymphoid units in glioblastoma.
glioblastoma or nonmalignant intracranial disease to report that the cranial