25 results for “huntingtons”. Showing 25 of 39,449.
Huntington's disease.
Huntington's disease, with particular focus on clinical characterization of prodromal
Imbalanced basal ganglia connectivity is associated with motor deficits and apathy in Huntington’s disease
Huntington’s disease, the preferential early loss of striatal neurons
Therapeutic reversal of Huntington's disease by in vivo self-assembled siRNAs.
Huntington's disease is an autosomal-dominant neurodegenerative disease caused
Reactive astrocytes promote proteostasis in Huntington's disease through the JAK2-STAT3 pathway.
Huntington's disease is a fatal neurodegenerative disease characterized by striatal
Shared patterns of glial transcriptional dysregulation link Huntington's disease and schizophrenia.
Huntington's disease and juvenile-onset schizophrenia have long been
Huntington's disease: from molecular pathogenesis to clinical treatment.
Huntington's disease is a progressive, fatal, neurodegenerative disorder caused
In vivo CRISPR-Cas9 genome editing in mice identifies genetic modifiers of somatic CAG repeat instability in Huntington's disease.
Huntington's disease, one of more than 50 inherited repeat
Sirt1 mediates neuroprotection from mutant huntingtin by activation of the TORC1 and CREB transcriptional pathway.
Huntington's disease, an inherited neurodegenerative disorder caused by a glutamine
Reduced Expression of Foxp1 as a Contributing Factor in Huntington's Disease.
Huntington's disease (HD) is an inherited neurodegenerative disease caused
Targeting Huntingtin Expression in Patients with Huntington's Disease.
Huntington's disease is an autosomal-dominant neurodegenerative disease caused
Potential disease-modifying therapies for Huntington's disease: lessons learned and future opportunities.
Huntington's disease is the most frequent autosomal dominant neurodegenerative
From Pathogenesis to Therapeutics: A Review of 150 Years of Huntington's Disease Research.
Huntington's disease (HD) is a debilitating neurodegenerative genetic disorder
Longitudinal modeling of human neuronal aging reveals the contribution of the RCAN1-TFEB pathway to Huntington's disease neurodegeneration.
Huntington's disease identified pathways involving RCAN1, a negative regulator
Huntington disease: somatic expansion, pathobiology and therapeutics.
Huntington disease is a fatal, inherited, neurodegenerative disease caused by a CAG repeat
Subtyping monogenic disorders: Huntington disease.
Huntington disease is a highly disabling neurodegenerative disease characterized by psychiatric
Huntington disease.
Huntington disease is devastating to patients and their families - with
Single-nucleus transcriptomics of an engineered pig model reveals microglia-T cell interactions driving Huntington's disease neurodegeneration.
Huntington's disease remains unresolved. Addressing this question requires models
Decoding Non-Neuronal Mechanisms and Therapeutic Targets in Huntington's Disease Through Integrative Transcriptomics and Machine Learning.
Huntington's disease (HD) is a rare, inherited neurodegenerative disorder
TYROBP/DAP12 knockout in Huntington's disease Q175 mice cell-autonomously decreases microglial expression of disease-associated genes and non-cell-autonomously mitigates astrogliosis and motor deterioration.
Huntington's disease (HD) is a fatal neurodegenerative disorder caused
Mutant HTT (huntingtin) impairs mitophagy in a cellular model of Huntington disease.
Huntington disease.Abbreviations: AMPK: AMP-activated protein kinase; ATG13: autophagy related
Aloe Polysaccharides against 3-Nitropropionic acid-induced Huntington's disease-like symptoms: Role of BDNF/NF-κB/Nrf2 signaling pathways.
Huntington's Disease (HD) is a neurodegenerative ailment characterized by progressive
Roflumilast Elicits Therapeutic and Neuroprotective Effects in 3-Nitropropionic Acid-Induced Huntington's Disease-Like Neurodegeneration in Rats by Mitigating NLRP3 Inflammasome-Mediated Pyroptosis, Ferroptosis, and Glial Activation.
Huntington’s disease (HD) pathogenesis involves diverse cellular mechanisms, yet the contributions
miR-34a-5p as molecular hub of pathomechanisms in Huntington's disease.
Huntington's disease (HD) is increasingly recognized, the molecular functions
Artemisinin attenuates 3-nitropropionic acid-induced neurodegeneration via HMGB1/TLR4/NF-κB modulation in a rat model of huntington's disease.
Huntington's disease (HD) is a progressive neurodegenerative disorder characterized
The roles of HSP40/DNAJ protein family in neurodegenerative diseases.
Huntington's disease, spinocerebellar ataxia, Charcot-Marie-Tooth disease, spinal