25 results for “late”. Showing 25 of 39,449.
10.1093/brain/awz099
LATE). LATE neuropathological change (LATE-NC) is defined by a stereotypical
Clinical criteria for limbic-predominant age-related TDP-43 encephalopathy.
late life. LATE neuropathologic change (LATE-NC) is a common
Pure LATE-NC: Frequency, clinical impact, and the importance of considering APOE genotype when assessing this and other subtypes of non-Alzheimer's pathologies.
LATE-NC cases, there was a trend for higher LATE
TDP-43 pathology is associated with increased tau burdens and seeding.
LATE-NC, AD(LATE-NC+), have increased burdens of pretangles
Limbic-predominant age-related TDP43 encephalopathy (LATE) neuropathological change in neurodegenerative diseases.
LATE, referred to as LATE neuropathological change (LATE-NC), consist
Predictors and long-term prognosis of early and late recurrence for patients undergoing hepatic resection of hepatocellular carcinoma: a large-scale multicenter study.
late recurrence (>2 years after surgery). Predictors of early and late
Utility of the Amygdalar Atrophy Scale to Identify Patients With Limbic-Predominant Age-Related TDP-43 Encephalopathy in a Memory Clinic.
LATE) is a neurodegenerative condition often overlapping clinically with Alzheimer
Distinct characteristics of amyloid deposits in early- and late-onset transthyretin Val30Met familial amyloid polyneuropathy.
late-onset cases. Amyloid deposition in late-onset cases may be similar
Data-driven neuropathological staging and subtyping of TDP-43 proteinopathies.
late-stage LATE-NC and early-stage FTLD-TDP. We further
Alzheimer's disease and its co-pathologies: Implications for hippocampal degeneration, cognitive decline, and the role of APOE ε4.
LATE-NC), α-synuclein, other age-related lesions, and apolipoprotein
Late effects after allogeneic haematopoietic cell transplantation in children and adolescents with non-malignant disorders: a retrospective cohort study.
late occurring toxic effects remain a challenge. We investigated the incidence
Limbic-predominant age-related TDP-43 encephalopathy (LATE-NC): Co-pathologies and genetic risk factors provide clues about pathogenesis.
LATE-NC is not rare, LATE-NC often coexists in the same
Changes in Alzheimer Disease Blood Biomarkers and Associations With Incident All-Cause Dementia.
late life had statistically significant associations with late-life dementia
Route of steroid-activated macromolecules through nuclear pores imaged with atomic force microscopy.
late (20 min) peak. Electrical peaks reflect macromolecule interaction with
Cooling for newborns with hypoxic ischaemic encephalopathy.
late preterm newborns with hypoxic ischaemic encephalopathy were identified by searching
Multivariate and Cladistic Analyses of Isolated Teeth Reveal Sympatry of Theropod Dinosaurs in the Late Jurassic of Northern Germany.
Late Jurassic theropod teeth are described of which the majority
No association between TGFB1 polymorphisms and late radiotherapy toxicity: a meta-analysis.
late radiation-induced injury of normal tissue, but the conclusions
Posttransplantation late complications increase over time for patients with SCID: A Primary Immune Deficiency Treatment Consortium (PIDTC) landmark study
late death (hazard ratio, 7.21; 95% confidence interval, 2.71-19.18; P < .001). Late
Author response: Cortico-thalamo-cortical interactions modulate electrically evoked EEG responses in mice
late component in the evoked EEG. We infer that cortico
Prevalence and factors associated with age-related macular degeneration in a southwestern island population of Japan: the Kumejima Study.
late AMD. Age-adjusted prevalence was 13.4% for any AMD, 13.3% for early
Glycidol induces axonopathy and aberrations of hippocampal neurogenesis affecting late-stage differentiation by exposure to rats in a framework of 28-day toxicity study.
late-stage neurogenesis in the hippocampal dentate gyrus of rat offspring
Regional interneuron transcriptional changes reveal pathologic markers of disease progression in a mouse model of Alzheimer's disease.
late-stage (30 weeks-of-age) disease. Global comparison of differentially
Rare variants in APP, PSEN1 and PSEN2 increase risk for AD in late-onset Alzheimer's disease families.
late onset AD (LOAD). Similarly, studies in single families have
Inhibition of miR-331-3p and miR-9-5p ameliorates Alzheimer's disease by enhancing autophagy.
late-stage (12 months) APPswe/PS1dE9 mice (hereinafter referred to as AD mice
Gene dose of apolipoprotein E type 4 allele and the risk of Alzheimer's disease in late onset families.
late onset familial and sporadic forms of Alzheimer's disease