25 results for “leukemia”. Showing 25 of 39,449.
Trans-activator of transcription-pre-B-cell leukemia transcription factor 1 alleviates Alzheimer's disease by reducing neuronal insulin resistance and restoring energy homeostasis.
leukemia transcription factor 1 as a crucial regulator of brain
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Leukemia (AML) in adults is a clinically devastating disease with
Detection of minimal residual disease in NPM1-mutated acute myeloid leukemia by next-generation sequencing
leukemia. Currently, minimal residual disease may be detected by RQ-PCR or flow
A CEBPB/IL-1β/TNF-α feedback loop drives drug resistance to venetoclax and MDM2 inhibitors in monocytic leukemia.
leukemia (AML) with wild-type TP53. Through an integrated analysis
Abstract PR004: High-density CRISPR screens reveal mechanisms of chromatin regulation of stemness networks in acute myeloid leukemia
leukemia-associated oncoproteins activate transcriptional circuits resembling a stem-like
Investigation on the mechanism of androsta-4,6,8,14-tetraene-3,11,16-trione against acute lymphoblastic leukemia.
leukemia agents, and Epigynum auritum is a Yunnan folk medicine
C1Q labels a highly aggressive macrophage-like leukemia population indicating extramedullary infiltration and relapse.
leukemia cutis. A complement C1Q+ macrophage-like leukemia subset, which
--Immunophenotypic Aberrancies in Acute Leukemia: A Tertiary Care Centre Experience.
leukemia (AML), 43 as acute lymphoid leukemia (ALL), and four
Bi-functional CpG-STAT3 decoy oligonucleotide triggers multilineage differentiation of acute myeloid leukemia in mice.
leukemia (AML) cells resist differentiation stimuli despite high expression of innate
Cytotoxic and antiproliferative activities of amphibian (anuran) skin extracts on human acute monocytic leukemia cells.
leukemia is the subtype M5 of acute myeloid leukemia (AML) a cancer
Targeting the Menin-KMT2A Axis in Acute Leukemia: From Epigenetic Dependency to Clinical Translation.
leukemias characterized by a shared epigenetic dependency on the menin
BRAF Mutations in Myeloid Neoplasms: Prevalence, Co-Mutation Landscape, and Clinical Outcomes-A Comprehensive Review.
leukemia and therapy-related or secondary acute myeloid leukemia. Both
Menin Inhibition With Revumenib for KMT2A-Rearranged Relapsed or Refractory Acute Leukemia (AUGMENT-101).
leukemia. Here, we evaluated the activity of revumenib in individuals
Genetic Inactivation of CD33 in Hematopoietic Stem Cells to Enable CAR T Cell Immunotherapy for Acute Myeloid Leukemia.
leukemia (AML) results in toxicity from destruction of normal myeloid
Systemic mastocytosis in the elderly.
leukemia (n = 7), acute myelocytic leukemia (n = 1), myelodysplastic syndrome
Evaluation of cationic channel TRPV2 as a novel biomarker and therapeutic target in Leukemia-Implications concerning the resolution of pulmonary inflammation.
leukemia face the risk of complications including pulmonary dysfunction that
Memory T cells from minor histocompatibility antigen–vaccinated and virus-immune donors improve GVL and immune reconstitution
leukemia cells, mediating the GVL effect. Donor T cells, including
High Density Domain-Focused CRISPR Screens Reveal Novel Epigenetic Regulators of <i>HOX/MEIS</i> Activation in Acute Myeloid Leukemia
leukemia (AML) and is linked to leukemic self-renewal and therapy
Precision medicine treatment in acute myeloid leukemia using prospective genomic profiling: feasibility and preliminary efficacy of the Beat AML Master Trial.
leukemia (AML) is the most common diagnosed leukemia. In older
Targetable kinase-activating lesions in Ph-like acute lymphoblastic leukemia.
leukemia (Ph-like ALL) is characterized by a gene-expression
CD96 as a therapeutic target for CAR T cell therapy in acute myeloid leukemia.
leukemia (AML) are limited by the scarcity of leukemia stem
The role of CD33 as therapeutic target in acute myeloid leukemia.
leukemia (AML) blasts in most patients and, possibly, leukemic stem
Cord blood CD8+ T-cell expansion following granulocyte transfusions eradicates refractory leukemia.
leukemia is principally mediated by donor T cells directed against
Dual-Specificity, Tyrosine Phosphorylation-Regulated Kinases (DYRKs) and cdc2-Like Kinases (CLKs) in Human Disease, an Overview.
leukemias (acute lymphoblastic leukemia, acute megakaryoblastic leukemia), viral infections (influenza
CX43-mediated mitochondrial transfer maintains stemness of KG-1a leukemia stem cells through metabolic remodeling.
leukemia (AML) is characterized by abundant immature myeloid cells, relapse