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4 results for “lrrk2-g2019s”. Showing 4 of 39,449.

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Nuclear ASC speck formation in microglia is associated with inflammasome priming and is exacerbated in LRRK2-G2019S Parkinson's disease.

G2019S mutation, enhance inflammatory signalling in microglia and peripheral immune cells. However, how LRRK2 intersects with the NLRP3 pathway remains unclear. Here, we investigate the role of
The Parkinson's-disease-associated mutation LRRK2-G2019S alters dopaminergic differentiation dynamics via NR2F1.

LRRK2-G2019S mutation alters the initial phase of neuronal differentiation
Genetic evidence for a functional association between Parkinson's disease proteins leucine-rich repeat kinase 2 and α-synuclein during axonal transport.

LRRK2-G2019S disrupts the axonal transport of autophagic vesicles and mitochondria
RAB12-LRRK2 complex suppresses primary ciliogenesis and regulates centrosome homeostasis in astrocytes.

LRRK2-G2019S mutation are prevented by Rab12 deletion in astrocytes

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Nuclear ASC speck formation in microglia is associated with inflammasome priming and is exacerbated in LRRK2-G2019S Parkinson's disease.

The Parkinson's-disease-associated mutation LRRK2-G2019S alters dopaminergic differentiation dynamics via NR2F1.

Genetic evidence for a functional association between Parkinson's disease proteins leucine-rich repeat kinase 2 and α-synuclein during axonal transport.

RAB12-LRRK2 complex suppresses primary ciliogenesis and regulates centrosome homeostasis in astrocytes.