25 results for “mogad”. Showing 25 of 39,449.
Longitudinal Retinal Changes in MOGAD.
MOGAD-ON) compared with MOGAD eyes without a history of ON (MOGAD
Diagnostic Value of the Kappa Free Light Chain Index to Distinguish MOGAD, NMOSD, and MS.
MOGAD), and multiple sclerosis (MS) can be complex. Kappa free
Granulocyte and astrocyte markers distinguish MOG-antibody disease and neuromyelitis optica from multiple sclerosis.
MOGAD) is less clear, and their usefulness for diagnostic differentiation
Psychiatric comorbidities cluster early after onset in MOGAD: a cross-sectional comparative study with MS and NMOSD.
MOGAD) are not well defined. We aimed to characterise the spectrum
Ganglion Cell Layer Compared With Inner Plexiform Layer Atrophy After Optic Neuritis Associated With NMOSD, MOGAD, and MS.
MOGAD). Compared with MS-ON, AQP4-ON and MOGAD-ON eyes
Complement Activation Profiles Predict Clinical Outcomes in Myelin Oligodendrocyte Glycoprotein Antibody-Associated Disease.
MOGAD. METHODS: Multicentric retrospective cohort study including patients with MOGAD
TRUE-MOGAD Score: A Novel Scoring System to Identify MOGAD Among Positive MOG-IgG Test Results.
MOGAD score is a practical tool that accurately identifies MOGAD
MOG CNS Autoimmunity and MOGAD.
MOGAD) is a distinct entity from either NMO or multiple
NMOSD and MOGAD: an evolving disease spectrum.
MOGAD and double-seronegative NMOSD. MOGAD shares features with NMOSD
Myelin Oligodendrocyte Glycoprotein Antibody-Associated Disease.
MOGAD). LATEST DEVELOPMENTS: The discovery of the MOG antibody as a diagnostic
Characteristics of MRI lesions in AQP4 antibody-positive NMOSD, MOGAD, and multiple sclerosis: a systematic review and meta-analysis.
MOGAD, and multiple sclerosis: a systematic review and meta-analysis
MRI to differentiate multiple sclerosis, neuromyelitis optica, and myelin oligodendrocyte glycoprotein antibody disease.
MOGAD) is crucial in clinical practice. The differential diagnosis may be challenging
MOGAD update: new phenotypes in the expanding clinical spectrum.
MOGAD) is often heralded by established clinical features, including optic
Cerebrospinal 14-3-3 Protein Levels as a Neuroaxonal Biomarker in Aquaporin-4 Antibody-Positive Neuromyelitis Optica Spectrum Disorder.
MOGAD) and multiple sclerosis (MS) and the association of CSF 14-3-3 protein
NMOSD and MOGAD.
MOGAD). The main differences between these disorders and multiple sclerosis
Therapeutic updates in NMOSD and MOGAD: From present practice to future promise.
MOGAD) are rare, antibody-mediated inflammatory disorders of the central
The pathology of central nervous system inflammatory demyelinating disease accompanying myelin oligodendrocyte glycoprotein autoantibody.
MOGAD. MOGAD pathology is dominated by coexistence of both perivenous
Diagnosis of myelin oligodendrocyte glycoprotein antibody-associated disease: International MOGAD Panel proposed criteria.
MOGAD) in which the presence of MOG-IgG is a core
Updates in NMOSD and MOGAD Diagnosis and Treatment: A Tale of Two Central Nervous System Autoimmune Inflammatory Disorders.
MOGAD) are antibody-associated diseases targeting astrocytes and oligodendrocytes, respectively
Blood-Brain Barrier Disruption in Neuroimmunological Disease.
MOGAD), autoimmune encephalitis (AE), and paraneoplastic neurological syndrome (PNS). The transmigration
Pediatric inflammatory leukoencephalopathies.
MOGAD) and aquaporin 4-antibody-positive neuromyelitis optica spectrum disorder
Overlapping autoimmunity and demyelination syndromes associated with TNF inhibitor therapy.
MOGAD) is a demyelinating disorder of the central nervous system
NMOSD and MOGAD: Updates on diagnostic criteria.
MOGAD) criteria have been validated in different populations, and a clear
Modeling MOG Antibody-Associated Disorder and Neuromyelitis Optica Spectrum Disorder in Animal Models: Visual System Manifestations.
MOGAD) are incompletely understood. The respective impact of optic nerve
Understanding Further the Phenotypic Spectrum of Central Nervous System Inflammatory Demyelinating Disorders Using Unsupervised Clustering.
MOGAD), occasionally overlap. Some patients remain double-seronegative, showing atypical