25 results for “Cerebrovascular Pathology”. Showing 25 of 39,449.
White matter hyperintensities and the risk of vascular dementia: a systematic review and meta-analysis.
cerebrovascular pathology. Some studies suggest that vascular hemodynamic changes contribute
Vascular cognitive impairment and dementia: Mechanisms, treatment, and future directions.
cerebrovascular pathology, while perhaps another 20% of vascular diseases contribute
Association of Vascular Risk Factors and Cerebrovascular Pathology With Alzheimer Disease Pathologic Changes in Individuals Without Dementia.
cerebrovascular pathology as key biomarkers for accurate design of anti
APOE, vascular pathology, and the AD brain.
pathology, adjusting for age at death, sex, brain weight, and Braak stage, were evaluated. APOE-epsilon4 was not associated with gross cerebrovascular
Cerebrovascular disease and dementia.
cerebrovascular pathology that is validated against increasing cognitive impairment. Although
Small vessel disease and Alzheimer's dementia: pathological considerations.
cerebrovascular pathology, which constitutes distinct small vessel disease (SVD). Cerebral
Neurodegeneration: 2024 update.
cerebrovascular pathologies over 25 years, associations between RHI and TDP-43 / hippocampal
APOE4-driven lipid metabolic dysregulation in Alzheimer's disease: Multi-pathway mechanisms and therapeutic perspectives.
cerebrovascular pathology. This article summarized the evidence that APOE4 affects
Iron induces blood-brain barrier alteration contributing to cognitive impairment in β-thalassaemia mice.
cerebrovascular pathology remain unclear. This study investigated the effects of heavy
Vascular Cognitive Impairment (VCI).
cerebrovascular pathology is the most important contributor to dementia, with
Vascular dementia.
cerebrovascular pathology and cognitive impairment, and the paucity of identifiable
Antiretroviral drugs efavirenz, dolutegravir and bictegravir dysregulate blood-brain barrier integrity and function.
cerebrovascular pathology, which may contribute to the associated short-term
Mechanisms of vascular disease in dementia: what does industry want to know?
cerebrovascular pathologies as these will maximize the opportunity of bringing
Biofluid biomarkers in Alzheimer's disease and other neurodegenerative dementias.
pathology, synaptic pathophysiology and cerebrovascular disease-pathologies and processes that
Sex and gender differences in cognitive resilience to aging and Alzheimer's disease.
pathologies (e.g., cerebrovascular disease) which ultimately shape cognitive trajectories. Understanding
Diabetes-Related Dementia.
pathology or cerebrovascular diseases. This type of dementia, not showing
Inhibition of IFITM3 in cerebrovascular endothelium alleviates Alzheimer's-related phenotypes.
cerebrovascular walls, reduced Alzheimer's-related pathology, and improved cognitive
Apolipoprotein E in Alzheimer's disease: molecular insights and therapeutic opportunities.
pathology, neuroinflammation, autophagy, cerebrovascular integrity and protection from lipid peroxidation
Vascular Heparan Sulfate and Amyloid-β in Alzheimer's Disease Patients.
cerebrovascular HS levels in AD patients with severe CAA. Overall, these findings reveal potential intricate interplay between HS, Aβ, ApoE, and vascular pathology
Aggregate effects of vascular risk factors on cerebrovascular changes in autopsy-confirmed Alzheimer's disease.
cerebrovascular and Alzheimer's disease (AD) pathologies. Eighty-four AD patients
Glucagon-like peptide-1 receptor agonists for major neurocognitive disorders.
pathological protein aggregates, proteostasis, cerebrovascular system and blood-brain barrier
Monocyte-derived IL-6 programs microglia to rebuild damaged brain vasculature.
Cerebrovascular injury (CVI) is a common pathology caused by infections
Blood and brain transcriptome analysis reveals APOE genotype-mediated and immune-related pathways involved in Alzheimer disease.
pathology. The APOE ε4 allele has shown cerebrovascular effects including
Pathologic and clinical correlates of region-specific brain GFAP in Alzheimer's disease.
pathology (amyloid-β and tau), cerebrovascular (microinfarcts, macroinfarcts, and cerebral
VSIG4 Attenuates NLRP3 and Ameliorates Neuroinflammation via JAK2-STAT3-A20 Pathway after Intracerebral Hemorrhage in Mice.
cerebrovascular disease. Neuroinflammation plays an important pathological role in brain