Upstream targets
Entities that most often appear as the source end
of a causal edge in substrate_causal_effects. The more
mechanisms flow out of an entity, the more upstream it is.
Companion view: /targets.
- #1 TDP-43 nuclear loss-of-function mechanism 4 edges
- #2 mitochondrial dysfunction mechanism 3 edges
- #3 CD8+ T cell recruitment mechanism 2 edges
- #4 multiplexed base editing mechanism 2 edges
- #5 oligodendrocyte sulfatide deficiency mechanism 2 edges
- #6 TDP-43 cytoplasmic aggregation mechanism 2 edges
- #7 aging mechanism 1 edge
- #8 AP1S1 downregulation mechanism 1 edge
- #9 APOE4 overexpression mechanism 1 edge
- #10 APOE4 removal mechanism 1 edge
- #11 astrocyte-derived inflammatory signals mechanism 1 edge
- #12 astrocyte-specific APOE4 knockout mechanism 1 edge
- #13 autophagy upregulation mechanism 1 edge
- #14 brain-derived exosomes from aged mice mechanism 1 edge
- #15 CAG repeat expansion reduction mechanism 1 edge
- #16 cellular senescence mechanism 1 edge
- #17 chronic hypoperfusion mechanism 1 edge
- #18 complement activation mechanism 1 edge
- #19 complex I deficiency mechanism 1 edge
- #20 CRISPRa with chromatin modifiers mechanism 1 edge
- #21 CRISPRi downregulation of MSH3 mechanism 1 edge
- #22 cytokine secretion mechanism 1 edge
- #23 cytoskeletal checkpoint disruption mechanism 1 edge