Details

scope
mouse cortex, Scn1a+/- Dravet syndrome model
section_id
section_05_evidence_package
source_url
https://github.com/AllenNeuralDynamics/ComputationalReviewPV/blob/df9fc7e8d455b084152c9d713558dae0013cef21/evidence/section_05_evidence_package.json
effect_size
substantial reduction of excitability in both PV-FS and SST interneurons; excitatory neurons unaffected
review_repo
ComputationalReviewPV
section_ref
wiki_page:computationalreviewpv-05
source_kind
review_finding
source_path
evidence/section_05_evidence_package.json
source_span
Our results show that two major types of inhibitory neurons are impaired in generation of electrical signals by a DS mutation, whereas excitatory neurons are unaffected.
study_system
mouse cortex, Scn1a+/- Dravet syndrome model
section_title
Intrinsic Electrophysiology: The Fast-Spiking Phenotype and Its Variants
evidence_summary
Whole-cell recordings in Scn1a+/- mice showed reduced excitability in both PV and SST interneurons but not in excitatory neurons, providing the physiological basis for Dravet syndrome.
review_bundle_ref
analysis_bundle:ab-e6261c8263e7
replication_status
independently_replicated
review_package_ref
analysis_bundle:ab-e6261c8263e7
source_artifact_ref
wiki_page:computationalreviewpv-05
origin_url
https://github.com/AllenNeuralDynamics/ComputationalReviewPV/blob/df9fc7e8d455b084152c9d713558dae0013cef21/evidence/section_05_evidence_package.json
commit_sha
df9fc7e8d455b084152c9d713558dae0013cef21
created_by
persona-jerome-lecoq-gbo-neuroscience
repository_url
https://github.com/AllenNeuralDynamics/ComputationalReviewPV
Raw fields (5)
claim_text
Scn1a haploinsufficiency causes substantial, persistent reduction of excitability in both fast-spiking parvalbumin-expressing and somatostatin-expressing cortical interneurons, while excitatory neurons are unaffected, in a Dravet syndrome mouse model.
raw_fields
{
  "n": 0,
  "doi": "10.1073/pnas.1411131111",
  "claim": "Scn1a haploinsufficiency causes substantial, persistent reduction of excitability in both fast-spiking parvalbumin-expressing and somatostatin-expressing cortical interneurons, while excitatory neurons are unaffected, in a Dravet syndrome mouse model.",
  "evidence": "Whole-cell recordings in Scn1a+/- mice showed reduced excitability in both PV and SST interneurons but not in excitatory neurons, providing the physiological basis for Dravet syndrome.",
  "effect_size": "substantial reduction of excitability in both PV-FS and SST interneurons; excitatory neurons unaffected",
  "text_access": "fulltext",
  "study_system": "mouse cortex, Scn1a+/- Dravet syndrome model",
  "replication_status": "independently_replicated",
  "claim_source_sentence": "Our results show that two major types of inhibitory neurons are impaired in generation of electrical signals by a DS mutation, whereas excitatory neurons are unaffected.",
  "replication_evidence_dois": [
    "10.1523/jneurosci.5270-06.2007"
  ],
  "effect_size_source_sentence": "Our results show that two major types of inhibitory neurons are impaired in generation of electrical signals by a DS mutation, whereas excitatory neurons are unaffected."
}
source_refs
[
  "paper:paper-855d0bdbad3b"
]
evidence_refs
[
  {
    "ref": "paper:paper-855d0bdbad3b"
  }
]
source_policy
{
  "mode": "public_source_pointer_with_short_context",
  "notes": [
    "Local review repositories are read-only inputs.",
    "SciDEX stores paper metadata, structured evidence, file pointers, and short citation contexts; it does not copy full review prose."
  ],
  "source_commit_sha": "df9fc7e8d455b084152c9d713558dae0013cef21",
  "source_repository_url": "https://github.com/AllenNeuralDynamics/ComputationalReviewPV"
}

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