Details

scope
mouse Scn1a+/- Dravet syndrome model
section_id
section_05_evidence_package
source_url
https://github.com/AllenNeuralDynamics/ComputationalReviewPV/blob/df9fc7e8d455b084152c9d713558dae0013cef21/evidence/section_05_evidence_package.json
effect_size
>1000-fold selectivity for Nav1.1; significant reduction in seizures and mortality
review_repo
ComputationalReviewPV
section_ref
wiki_page:computationalreviewpv-05
source_kind
review_finding
source_path
evidence/section_05_evidence_package.json
source_span
Utilizing a mouse model of Dravet syndrome, Hm1a restored inhibitory neuron function and significantly reduced seizures and mortality in heterozygote mice.
study_system
mouse Scn1a+/- Dravet syndrome model
section_title
Intrinsic Electrophysiology: The Fast-Spiking Phenotype and Its Variants
evidence_summary
Hm1a showed >1000-fold selectivity for hNav1.1 over other Nav1.x members. Mechanism involves slowing of channel inactivation. Proof-of-concept for targeted therapy in genetic epilepsy.
review_bundle_ref
analysis_bundle:ab-e6261c8263e7
replication_status
independently_replicated
review_package_ref
analysis_bundle:ab-e6261c8263e7
source_artifact_ref
wiki_page:computationalreviewpv-05
origin_url
https://github.com/AllenNeuralDynamics/ComputationalReviewPV/blob/df9fc7e8d455b084152c9d713558dae0013cef21/evidence/section_05_evidence_package.json
commit_sha
df9fc7e8d455b084152c9d713558dae0013cef21
created_by
persona-jerome-lecoq-gbo-neuroscience
repository_url
https://github.com/AllenNeuralDynamics/ComputationalReviewPV
Raw fields (5)
claim_text
Selective pharmacological activation of Nav1.1 with the spider venom peptide Hm1a restores inhibitory neuron function and significantly reduces seizures and mortality in Scn1a+/- Dravet syndrome mice, demonstrating Nav1.1 as a therapeutic target.
raw_fields
{
  "n": 0,
  "doi": "10.1073/pnas.1804764115",
  "claim": "Selective pharmacological activation of Nav1.1 with the spider venom peptide Hm1a restores inhibitory neuron function and significantly reduces seizures and mortality in Scn1a+/- Dravet syndrome mice, demonstrating Nav1.1 as a therapeutic target.",
  "evidence": "Hm1a showed >1000-fold selectivity for hNav1.1 over other Nav1.x members. Mechanism involves slowing of channel inactivation. Proof-of-concept for targeted therapy in genetic epilepsy.",
  "effect_size": ">1000-fold selectivity for Nav1.1; significant reduction in seizures and mortality",
  "text_access": "fulltext",
  "study_system": "mouse Scn1a+/- Dravet syndrome model",
  "replication_status": "independently_replicated",
  "claim_source_sentence": "Utilizing a mouse model of Dravet syndrome, Hm1a restored inhibitory neuron function and significantly reduced seizures and mortality in heterozygote mice.",
  "replication_evidence_dois": [
    "10.1016/j.molpha.2025.100072"
  ],
  "effect_size_source_sentence": "Hm1a restored inhibitory neuron function and significantly reduced seizures and mortality in heterozygote mice."
}
source_refs
[
  "paper:paper-4bb49760633b"
]
evidence_refs
[
  {
    "ref": "paper:paper-4bb49760633b"
  }
]
source_policy
{
  "mode": "public_source_pointer_with_short_context",
  "notes": [
    "Local review repositories are read-only inputs.",
    "SciDEX stores paper metadata, structured evidence, file pointers, and short citation contexts; it does not copy full review prose."
  ],
  "source_commit_sha": "df9fc7e8d455b084152c9d713558dae0013cef21",
  "source_repository_url": "https://github.com/AllenNeuralDynamics/ComputationalReviewPV"
}

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